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Combination treatment with triptolide and hydroxycamptothecin synergistically enhances apoptosis in A549 lung adenocarcinoma cells through PP2A-regulated ERK, p38 MAPKs and Akt signaling pathways

机译：用雷公藤苷和羟基清除素的组合治疗通过PP2A调节的ERK，P38 MAPKS和AKT信号通路协同增强A549肺腺癌细胞的细胞凋亡

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GUANMIN MENG; WEI WANG; KEQUN CHAI; SUWEN YANG; FANGQIONG LI; KAI JIANG;
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1. Combination treatment with triptolide and hydroxycamptothecin synergistically enhances apoptosis in A549 lung adenocarcinoma cells through PP2A-regulated ERK, p38 MAPKs and Akt signaling pathways [J] . Guanmin Meng, Wei Wang, Kequn Chai, International journal of oncology . 2015,第3期

机译：雷公藤内酯醇和羟基喜树碱联合治疗通过PP2A调控的ERK，p38 MAPKs和Akt信号通路协同增强A549肺腺癌细胞的凋亡
2. 2-Methoxy-1,4-naphthoquinone (MNQ) induces apoptosis of A549 lung adenocarcinoma cells via oxidation-triggered JNK and p38 MAPK signaling pathways [J] . Ong Jeremy Yee Hoong, Yong Phelim Voon Chen, Lim Yang Mooi, Life sciences . 2015,第Null期

机译：2-甲氧基-1,4-萘醌（MNQ）通过氧化触发的JNK和p38 MAPK信号通路诱导A549肺腺癌细胞凋亡
3. Combined Treatment with Triptolide and Tyrosine Kinase Inhibitors Synergistically Enhances Apoptosis in Non-small Cell Lung Cancer H1975 Cells but Not H1299 Cells through EGFR/Akt Pathway [J] . Xiaopei Tong, Pei Jiang, Yao Li, Chemical and Pharmaceutical Bulletin . 2019,第8期

机译：雷公藤内酯醇和酪氨酸激酶抑制剂的联合治疗通过EGFR / Akt途径协同增强非小细胞肺癌H1975细胞而非H1299细胞的凋亡
4. The Mechanism of Apoptosis in Adenocarcinoma of Lung Cancer A549 Cells Induced by Albumin-Derived from Peanut [C] . Minhui Long, Yuejun Sun, Ni Chen, International conference on applied biotechnology . 2014

机译：花生白蛋白诱导肺癌A549细胞凋亡的机制
5. MAPKs ERK, JNK and P38 in inflammatory and oxidative stress signaling investigated in human lung and skin fibroblasts. [D] . Mendonsa, Graziella. 2009

机译：在人肺和皮肤成纤维细胞中研究了炎症和氧化应激信号转导中的MAPK ERK，JNK和P38。
6. Combination treatment with triptolide and hydroxycamptothecin synergistically enhances apoptosis in A549 lung adenocarcinoma cells through PP2A-regulated ERK p38 MAPKs and Akt signaling pathways [O] . GUANMIN MENG, WEI WANG, KEQUN CHAI, -1

机译：雷公藤内酯醇和羟基喜树碱联合治疗通过PP2A调控的ERKp38 MAPKs和Akt信号通路协同增强A549肺腺癌细胞的凋亡
7. A Ferulic Acid Derivative FXS-3 Inhibits Proliferation and Metastasis of Human Lung Cancer A549 Cells via Positive JNK Signaling Pathway and Negative ERK/p38, AKT/mTOR and MEK/ERK Signaling Pathways [O] . Shi-Jun Yue, Peng-Xuan Zhang, Yue Zhu, 2019

机译：阿魏酸衍生物FXS-3通过阳性JNK信号通路和负ERK / P38，AKT / MTOR和MEK / ERK信号通路抑制人肺癌A549细胞的增殖和转移

Combination treatment with triptolide and hydroxycamptothecin synergistically enhances apoptosis in A549 lung adenocarcinoma cells through PP2A-regulated ERK, p38 MAPKs and Akt signaling pathways

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