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Allodynia and hyperalgesia in diabetic rats are mediated by GABA and depletion of spinal potassium-chloride co-transporters

机译：Allodynia和糖尿病大鼠的痛觉疗程由GABA和脊柱氯化钾共转运液的耗尽介导

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Corinne G. Jolivalt; Corinne A. Lee; Khara M. Ramos; Nigel A. Calcutt;
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1. Allodynia and hyperalgesia in diabetic rats are mediated by GABA and depletion of spinal potassium-chloride co-transporters. [J] . Jolivalt CG, Lee CA, Ramos KM, Pain. . 2008,第1期

机译：糖尿病大鼠的异常性疼痛和痛觉过敏是由GABA和脊髓氯化钾共转运蛋白的消耗介导的。
2. Spinal EP receptors mediating prostaglandin E2-induced mechanical hyperalgesia, thermal hyperalgesia, and touch-evoked allodynia in rats. [J] . Mebane H, Turnbach ME, Randich A The journal of pain: official journal of the American Pain Society . 2003,第7期

机译：脊髓EP受体介导前列腺素E 2诱导的大鼠机械性痛觉过敏，热痛觉过敏和触摸诱发的异常性疼痛。
3. Gabapentin reduces allodynia and hyperalgesia in painful diabetic neuropathy rats by decreasing expression level of Nav1.7 and p-ERK1/2 in DRG neurons [J] . ZhangJ.-L., YangJ.-P., ZhangJ.-R., Brain research . 2013,第Null期

机译：加巴喷丁通过降低DRG神经元中Nav1.7和p-ERK1 / 2的表达水平来减轻糖尿病性神经痛大鼠的异常性疼痛和痛觉过敏
4. Repeated whole-body vibration exposure induces prolonged mechanical hyperalgesia increased spinal COX-2: A novel rat model [C] . Guarino B. B., Baig H. A., Branconi J. L., Bioengineering Conference (NEBEC), 2012 38th Annual Northeast . 2012

机译：反复的全身振动暴露会导致长时间的机械性痛觉过敏和增加的脊髓COX-2：一种新型大鼠模型
5. The loss of GABAergic inhibitory interneurons correlates with the development of thermal hyperalgesia after spinal cord injury. [D] . Marsh, Adam. 2008

机译：GABA能抑制性中枢神经元的丧失与脊髓损伤后热痛觉过敏的发展有关。
6. Allodynia and hyperalgesia in diabetic rats are mediated by GABA and depletion of spinal potassium-chloride co-transporters [O] . Corinne G. Jolivalt, Corinne A. Lee, Khara M. Ramos, -1

机译：糖尿病大鼠的异常性疼痛和痛觉过敏由GABA和脊髓氯化钾共转运蛋白的耗竭介导
7. TRPV1 in GABAergic Interneurons Mediates Neuropathic Mechanical Allodynia and Disinhibition of the Nociceptive Circuitry in the Spinal Cord [O] . Kim Yong Ho, Back Seung Keun, Davies Alexander J., 2012

机译：GABA能中神经元中的TRPV1介导脊髓神经痛性机械性异常性疼痛和伤害感受电路的抑制作用。

Allodynia and hyperalgesia in diabetic rats are mediated by GABA and depletion of spinal potassium-chloride co-transporters

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