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Esa1, anArabidopsismutant with enhanced susceptibility to a range of necrotrophic fungal pathogens, shows a distorted induction of defense responses by reactive oxygen generating compounds

机译：ESA1，Anarabidopsicutants具有增强对一系列病症真菌病原体的易感性，表现出通过反应性氧生成化合物对防御反应的扭曲诱导

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Koenraad F. M.-J. Tierens; Bart P. H. J. Thomma; Rajendra P. Bari; Marie Garmier; Kristel Eggermont; Margreet Brouwer; Iris A. M. A. Penninckx; Willem F. Broekaert; Bruno P. A. Cammue;
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1. Plant Defense Signaling and Responses Against Necrotrophic Fungal Pathogens [J] . Pandey Dinesh, Rajendran Subin Raj Cheri Kunnumal, Gaur Manu, Journal of Plant Growth Regulation . 2016,第4期

机译：植物防御信号转导和对坏死性真菌病原体的反应。
2. Fight to the death: Arabidopsis thaliana defense response to fungal necrotrophic pathogens [J] . ?a?niewska Joanna, Macioszek Violetta K., Lawrence Christopher B., Acta Physiologiae Plantarum . 2010,第1期

机译：战斗致死：拟南芥对真菌坏死性病原体的防御反应
3. Fight to the death: Arabidopsis thaliana defense response to fungal necrotrophic pathogens [J] . Joanna Łaźniewska, Violetta K. Macioszek, Christopher B. Lawrence, Acta Physiologiae Plantarum . 2010,第1期

机译：战斗致死：拟南芥对真菌坏死性病原体的防御反应
4. The interaction of natural variation in a necrotrophic fungal pathogen, Botrytis cinerea, with plant defense in the Arabidopsis thaliana model system. [D] . Rowe, Heather Cristina. 2009

机译：在拟南芥模型系统中，坏死性真菌病原体灰葡萄孢中自然变异与植物防御的相互作用。
5. Pseudomonas fluorescens DN16 Enhances Cucumber Defense Responses Against the Necrotrophic Pathogen Botrytis cinerea by Regulating Thermospermine Catabolism [O] . Lin Zhu, Nana Qian, Yujun Sun, 2021

机译：通过调节热焦氨酸分解代谢荧光荧光荧光笔DN16增强了针对坏养殖病原体Botrytis inerea的黄瓜防御反应
6. The Arabidopsis ATAF1, a NAC Transcription Factor, Is a Negative Regulator of Defense Responses Against Necrotrophic Fungal and Bacterial Pathogens [O] . Xiaoe Wang, B. M. Vindhya S. Basnayake, Huijuan Zhang, 2009

机译：Arabidopsis Ataf1，NAC转录因子是对病症抗病性真菌和细菌病原体的防御反应的负调节因子

Esa1, anArabidopsismutant with enhanced susceptibility to a range of necrotrophic fungal pathogens, shows a distorted induction of defense responses by reactive oxygen generating compounds

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