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Interleukin-15 deficiency promotes the development of T-cell acute lymphoblastic leukemia in non-obese diabetes mice with severe combined immunodeficiency

机译：白细胞介素-15缺乏促进非肥胖糖尿病小鼠的T细胞急性淋巴细胞白血病的发育，具有严重的免疫缺陷

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D Bobbala; R Kandhi; X Chen; M Mayhue; E Bouchard; J Yan; H Knecht; F Barabé; S Ramanathan; S Ilangumaran;
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1. Antigen Loading (e.g., Glutamic Acid Decarboxylase 65) of Tolerogenic DCs (tolDCs) Reduces Their Capacity to Prevent Diabetes in the Non-Obese Diabetes (NOD)-Severe Combined Immunodeficiency Model of Adoptive Cotransfer of Diabetes As Well As in NOD Mice [J] . David P. Funda, Jaroslav Goliá?, Tomá? Hudcovic, Frontiers in Immunology . 2018,第1期

机译：致耐受性DC（tolDC）的抗原负荷（例如谷氨酸脱羧酶65）降低了其预防非肥胖糖尿病（NOD）-糖尿病和NOD小鼠过继共转移的严重联合免疫功能低下模型的能力。
2. Anti-CD19 immunotoxin enhances the activity of chemotherapy in severe combined immunodeficient mice with human pre-B acute lymphoblastic leukemia. [J] . Herrera L, Stanciu-Herrera C, Morgan C, Leukemia and lymphoma . 2006,第11期

机译：抗CD19免疫毒素可增强患有人B前急性淋巴细胞白血病的严重联合免疫缺陷小鼠的化疗活性。
3. 4134 Treatment of adolescents and young adults with T-cell acute lymphoblastic leukemia and lymphoblastic lymphoma according to the pediatric strategy for acute lymphoblastic leukemia - single center experience in Russia [J] . S.V.Semochkin, S.S.Kulikova, O.A.Kudryavceva, European Journal of Cancer Supplements . 2009,第2期

机译：4134根据急性淋巴细胞白血病的儿科策略治疗T细胞急性淋巴细胞白血病和淋巴母细胞淋巴瘤的青少年-俄罗斯的单中心经验
4. Creation of chemical compounds with higher growth inhibitoryactivity for T-cell acute lymphoblastic leukemia-derived cells [C] . Tomoya Higashi, ChihiroYoshida, Yoshifumi Hachiro 日本化学会春季年会 . 2020

机译：用于T细胞急性淋巴细胞白血病衍生细胞的较高生长抑制性的化合物的产生
5. Dissecting the Molecular Mechanism of TLX1-mediated T-cell Acute Lymphoblastic Leukemia Development Using an In Vitro Immortalization Model. [D] . Renn, Lynnsey Zweier. 2011

机译：使用体外永生化模型剖析TLX1介导的T细胞急性淋巴细胞白血病发展的分子机制。
6. Antigen Loading (e.g. Glutamic Acid Decarboxylase 65) of Tolerogenic DCs (tolDCs) Reduces Their Capacity to Prevent Diabetes in the Non-Obese Diabetes (NOD)-Severe Combined Immunodeficiency Model of Adoptive Cotransfer of Diabetes As Well As in NOD Mice [O] . David P. Funda, Jaroslav Goliáš, Tomáš Hudcovic, -1

机译：致耐受性DC（tolDC）的抗原负载（例如谷氨酸脱羧酶65）降低了其预防非肥胖糖尿病（NOD）-糖尿病和NOD小鼠过继转移的严重联合免疫功能低下模型的能力。
7. Growth of Human T-Cell Lineage Acute Leukemia in Severe Combined Immunodeficiency (SCID) Mice and Non-obese Diabetic SCID Mice [O] . Jeremy P.C. Steele, Robyn D. Clutterbuck, Ray L. Powles, 1997

机译：严重联合免疫缺陷（SCID）小鼠和非肥胖糖尿病SCID小鼠的人T细胞谱系急性白血病生长

Interleukin-15 deficiency promotes the development of T-cell acute lymphoblastic leukemia in non-obese diabetes mice with severe combined immunodeficiency

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