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Antiproliferative Effect of 1 ,25-Dihydroxyvitamin D3 in Human Prostate Cancer Cell Line LNCaP Involves Reduction of Cyclin-Dependent Kinase 2 Activity and Persistent G1 Accumulation

机译：人前列腺癌细胞系LNCAP中1,25-二羟基苯胺D3的抗增殖效应涉及细胞周期蛋白依赖性激酶2活性和持久性G1积累

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S.-H. Zhuang;
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1. Antiproliferative effect of 1α,25-dihydroxyvitamin D3 involves upregulation of cyclin-dependent kinase inhibitor p21 in human pancreatic cancer cells [J] . KanemaruM., MaeharaN., ChijiiwaK. Hepato-gastroenterology. . 2013,第125期

机译：1α，25-二羟基维生素D3的抗增殖作用涉及人胰腺癌细胞中细胞周期蛋白依赖性激酶抑制剂p21的上调
2. The Iroquois Homeobox Gene 5 Is Regulated by 1,25-Dihydroxyvitamin D3 in Human Prostate Cancer and Regulates Apoptosis and the Cell Cycle in LNCaP Prostate Cancer Cells. [J] . Myrthue A, Rademacher BL, Pittsenbarger J, Clinical cancer research: an official journal of the American Association for Cancer Research . 2008,第11期

机译：Iroquois Homeobox基因5在人前列腺癌中受1,25-二羟基维生素D3调节，并在LNCaP前列腺癌细胞中调节细胞凋亡和细胞周期。
3. 1alpha,25-Dihydroxyvitamin D3-3beta-(2)-bromoacetate, an affinity labeling derivative of 1alpha,25-dihydroxyvitamin D3 displays strong antiproliferative and cytotoxic behavior in prostate cancer cells. [J] . Swamy N, Persons KS, Chen TC, Journal of cellular biochemistry. . 2003,第5期

机译：1alpha，25-dihydroxyvitamin D3-3beta-（2）-溴乙酸盐，1alpha，25-dihydroxyvitamin D3的亲和标记衍生物在前列腺癌细胞中显示出强大的抗增殖和细胞毒性行为。
4. Black Tea Polyphenols Theaflavins Inhibit the Growth of LNCaP Prostate Cancer Cells through Suppressing Androgen Receptor and 5α-Reductase Activity [C] . Jen-Kun Lin, Hung-Hsiao Lee, Chi-Tang Ho Dietary supplements . -1

机译：红茶多酚茶黄素通过抑制雄激素受体和5α-还原酶的活性抑制LNCaP前列腺癌细胞的生长
5. Effect of androgen and natural compounds on proliferation of androgen-independent LNCaP human prostate cancer cells and prostate cancer progression. [D] . Chuu, Chih-pin. 2005

机译：雄激素和天然化合物对雄激素非依赖性LNCaP人前列腺癌细胞增殖和前列腺癌进展的影响。
6. Menthol Enhances an Antiproliferative Activity of 1α25-Dihydroxyvitamin D3 in LNCaP Cells [O] . Eun-Jung Park, Su-Hwa Kim, Byung-Joo Kim, 2009

机译：薄荷醇增强抗增殖活性LNCaP细胞中1α25-二羟基维生素D3的表达
7. p53 Is Required for 1,25-Dihydroxyvitamin D3-Induced G0 Arrest But Is Not Required for G1 Accumulation or Apoptosis of LNCaP Prostate Cancer Cells [O] . Tara C. Polek, LaMonica V. Stewart, Elizabeth J. Ryu, 2003

机译：P53是1,25-二羟基维胺D3诱导的G0停滞所必需的，但是LNCAP前列腺癌细胞的G1积累或凋亡不需要

Antiproliferative Effect of 1 ,25-Dihydroxyvitamin D3 in Human Prostate Cancer Cell Line LNCaP Involves Reduction of Cyclin-Dependent Kinase 2 Activity and Persistent G1 Accumulation

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