首页> 外文OA文献 >Cognitive Deficit in Amyloid-β–Injected Mice Was Improved by Pretreatment With a Low Dose of Telmisartan Partly Because of Peroxisome Proliferator-Activated Receptor-γ Activation

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Cognitive Deficit in Amyloid-β–Injected Mice Was Improved by Pretreatment With a Low Dose of Telmisartan Partly Because of Peroxisome Proliferator-Activated Receptor-γ Activation

机译：通过用低剂量的Telmisartan进行淀粉样蛋白-β-注入小鼠中的认知缺损，部分原因是由于过氧化物酶体增殖物激活的受体-γ活化

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Kana Tsukuda; Masaki Mogi; Jun Iwanami; Li-Juan Min; Akiko Sakata; Fei Jing; Masaru Iwai; Masatsugu Horiuchi;
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1. Cognitive Deficit in Amyloid-β–Injected Mice Was Improved by Pretreatment With a Low Dose of Telmisartan Partly Because of Peroxisome Proliferator-Activated Receptor-γ Activation [J] . Kana Tsukuda, Masaki Mogi, Jun Iwanami, Hypertension: An Official Journal of the American Heart Association . 2009,第4期

机译：低剂量替米沙坦预处理可改善β-淀粉样注射小鼠的认知缺陷，部分原因是过氧化物酶体增殖物激活受体-γ激活
2. Cognitive Deficit in Amyloid-beta-Injected Mice WasImproved by Pretreatment With a Low Dose of TelmisartanPartly Because of Peroxisome Proliferator-ActivatedReceptor-gamma Activation [J] . Masatsugu Horiuchi, Kana Tsukuda, Masaki Mogi Hypertension: An Official Journal of the American Heart Association . 2009,第4期

机译：低剂量替米沙坦预处理可改善淀粉样β注射小鼠的认知缺陷，部分原因是过氧化物酶体增殖物激活受体-γ激活
3. Nonhypotensive dose of telmisartan attenuates cognitive impairment partially due to peroxisome proliferator-activated receptor-gamma activation in mice with chronic cerebral hypoperfusion. [J] . Washida K, Ihara M, Nishio K, Stroke: A Journal of Cerebral Circulation . 2010,第8期

机译：替米沙坦的非低血压剂量可部分减轻过氧化物酶体增殖物激活的受体-γ在慢性脑灌注不足小鼠中的认知障碍。
4. INHIBITION OF THE EXPRESSION OF IgE HEAVY CHAIN GERMLINE TRANSCRIPTS BY PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR-γ LIGANDS [C] . Yoshiyuki MIYAZAKI, Hirofumi TACHIBANA, Koji YAMADA Annual Meeting of the Japanese Association for Animal Cell Technology . 2003

机译：过氧化物体增殖物激活受体-γ配体抑制IgE重链种系转录物的表达
5. Peroxisome Proliferator-activated Receptor-γ (PPARγ) Agonists: Potential Novel Treatment for HIV-1 Associated Brain Inflammation [D] . Omeragic, Amila . 2020

机译：过氧化物体增殖物激活受体-γ（PPARγ）激动剂：对HIV-1相关脑炎症的潜在新待遇
6. Mechanisms Underlying the Rapid Peroxisome Proliferator-Activated Receptor-γ-Mediated Amyloid Clearance and Reversal of Cognitive Deficits in a Murine Model of Alzheimers Disease [O] . Shweta Mandrekar-Colucci, J. Colleen Karlo, Gary E. Landreth 2012

机译：过氧化物酶体增殖物激活受体-γ介导的淀粉样蛋白清除和认知缺陷的逆转在阿尔茨海默氏病小鼠模型中的机制。
7. Selective peroxisome proliferator-activated receptor-α modulator K-877 efficiently activates the peroxisome proliferator-activated receptor-α pathway and improves lipid metabolism in mice [O] . Takei Kenta, Han Song-iee, Murayama Yuki, 2017

机译：选择性过氧化物酶体增殖物激活受体-α调节剂K-877有效激活过氧化物酶体增殖物激活受体-α途径并改善小鼠的脂质代谢

Cognitive Deficit in Amyloid-β–Injected Mice Was Improved by Pretreatment With a Low Dose of Telmisartan Partly Because of Peroxisome Proliferator-Activated Receptor-γ Activation

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