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Long-QT founder variant T309I-Kv7.1 with dominant negative pattern may predispose delayed afterdepolarizations under β-adrenergic stimulation

机译：Long-Qt创始人变体T309i-KV7.1具有显性负面图案可以在β-肾上腺素能刺激下易于延迟延迟后升级

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Iva Synková; Markéta Bébarová; Irena Andršová; Larisa Chmelikova; Olga Švecová; Jan Hošek; Michal Pásek; Pavel Vít; Iveta Valášková; Renata Gaillyová; Rostislav Navrátil; Tomáš Novotný;
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1. Block of I(Ks) does not induce early afterdepolarization activity but promotes beta-adrenergic agonist-induced delayed afterdepolarization activity. [J] . Burashnikov A, Antzelevitch C Journal of cardiovascular electrophysiology . 2000,第4期

机译：I（Ks）的阻滞不会诱导早期去极化活性，但会促进β-肾上腺素能激动剂诱导的延迟去极化活性。
2. A dominant RAD51C pathogenic splicing variant predisposes to breast and ovarian cancer in the Newfoundland population due to founder effect [J] . Lesa M. Dawson, Kerri N. Smith, Salem Werdyani, Molecular Genetics & Genomic Medicine . 2020,第2期

机译：由于创始效应，占牛蒡子中乳腺癌和卵巢癌的致辐射51C致病变异易感性
3. Role of α-Adrenergic Receptors in the Development of Delayed Afterdepolarization [J] . Kim Jae-Ha, Joo Kyung-Chae, Ju Jeong-Min, Korean Circulation Journal . 1996,第5期

机译：α-肾上腺素能受体在延迟后的作用中的作用
4. Differential responses to beta-adrenergic stimulation in the long-QT syndrome type 1: Characterization and mechanisms [C] . David Adolfo Sampedro-Puente, Jesus Fernandez-Bes, Esther Pueyo Computing in Cardiology Conference . 2017

机译：1型长QT综合征对β-肾上腺素能刺激的差异反应：表征和机制
5. A dominant RAD51C pathogenic splicing variant predisposes to breast and ovarian cancer in the Newfoundland population due to founder effect [O] . Lesa M. Dawson, Kerri N. Smith, Salem Werdyani, 2020

机译：RAD51C的主要致病性剪接变体由于创始人效应而易患纽芬兰人的乳腺癌和卵巢癌
6. Beta-adrenergic stimulation reverses the I Kr–I Ks dominant pattern during cardiac action potential [O] . Tamas Banyasz, Zhong Jian, Balazs Horvath, 2014

机译：β-肾上腺素能刺激在心脏动作潜力期间逆转I KR-I KS主导模式

Long-QT founder variant T309I-Kv7.1 with dominant negative pattern may predispose delayed afterdepolarizations under β-adrenergic stimulation

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