首页> 外文OA文献 >Promoter CpG island hypermethylation- and H3K9me3 and H3K27me3-mediated epigenetic silencing targets the deleted in colon cancer (DCC) gene in colorectal carcinogenesis without affecting neighboring genes on chromosomal region 18q21

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Promoter CpG island hypermethylation- and H3K9me3 and H3K27me3-mediated epigenetic silencing targets the deleted in colon cancer (DCC) gene in colorectal carcinogenesis without affecting neighboring genes on chromosomal region 18q21

机译：启动子CPG岛高甲基化 - 和H3K9ME3和H3K27ME3介导的表观遗传沉默靶向结肠癌中的结肠癌（DCC）基因的缺失，而不会影响染色体区域18Q21上的相邻基因

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Sarah Derks; Linda J.W. Bosch; Hanneke E.C. Niessen; Peter T.M. Moerkerk; Sandra M. van den Bosch; Beatriz Carvalho; Sandra Mongera; J.Willem Voncken; Gerrit A. Meijer; Adriaan P. de Bruïne; James G. Herman; Manon van Engeland;
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1. Promoter CpG island hypermethylation- and H3K9me3 and H3K27me3-mediated epigenetic silencing targets the deleted in colonn cancer (DCC) gene in colorectal carcinogenesis without affecting neighboring genes on chromosomal region 18q21 [J] . Manon van Engeland Carcinogenesis . 2009,第6期

机译：启动子CpG岛超甲基化和H3K9me3和H3K27me3介导的表观遗传沉默靶向结肠癌（DCC）基因在大肠癌发生中的缺失，而不影响染色体区域18q21上的邻近基因
2. Promoter CpG island hypermethylation- and H3K9me3 and H3K27me3-mediated epigenetic silencing targets the deleted in colon cancer (DCC) gene in colorectal carcinogenesis without affecting neighboring genes on chromosomal region 18q21. [J] . Derks S, Bosch LJ, Niessen HE Carcinogenesis . 2009,第6期

机译：启动子CpG岛高甲基化和H3K9me3和H3K27me3介导的表观遗传沉默靶向大肠癌发生中结肠癌（DCC）基因中的缺失，而不影响染色体区域18q21上的邻近基因。
3. Epigenetic silencing of microRNA-34b/c and B-cell translocation gene 4 is associated with CpG island methylation in colorectal cancer. [J] . Toyota M, Suzuki H, Sasaki Y, Cancer research: The official organ of the American Association for Cancer Research, Inc . 2008,第11期

机译：microRNA-34b / c和B细胞易位基因4的表观遗传沉默与结直肠癌中CpG岛甲基化有关。
4. Epigenetic regulation of non CpG island gene promoters. [D] . Cortez, Connie C. 2008

机译：非CpG岛基因启动子的表观遗传调控。
5. Dietary methyl donors methyl metabolizing enzymes and epigenetic regulators: diet–gene interactions and promoter CpG island hypermethylation in colorectal cancer [O] . Stefan de Vogel, Kim A. D. Wouters, Ralph W. H. Gottschalk, -1

机译：饮食中的甲基供体甲基代谢酶和表观遗传调节剂：大肠癌中饮食与基因的相互作用和启动子CpG岛超甲基化
6. Dietary methyl donors, methyl metabolizing enzymes, and epigenetic regulators: diet-gene interactions and promoter CpG island hypermethylation in colorectal cancer. [O] . de Vogel, S.C., Wouters, K.A., Gottschalk, R.W., 2011

机译：饮食中的甲基供体，甲基代谢酶和表观遗传调节剂：大肠癌中饮食与基因的相互作用以及启动子CpG岛超甲基化。

Promoter CpG island hypermethylation- and H3K9me3 and H3K27me3-mediated epigenetic silencing targets the deleted in colon cancer (DCC) gene in colorectal carcinogenesis without affecting neighboring genes on chromosomal region 18q21

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