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Secreted Form of Amyloid Precursor Protein Enhances Basal Glucose and Glutamate Transport and Protects Against Oxidative Impairment of Glucose and Glutamate Transport in Synaptosomes by a Cyclic GMP-Mediated Mechanism

机译：分泌形式的淀粉样蛋白前体蛋白质增强基底葡萄糖和谷氨酸转运，并通过环状的GMP介导的机制来保护血糖瘤和谷氨酸葡萄糖输送的氧化损伤

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Mark P. Mattson; Zhi Hong Guo; Jonathan D. Geiger;
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年度 2002
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1. Secreted form of amyloid precursor protein enhances basal glucose and glutamate transport and protects against oxidative impairment of glucose and glutamate transport in synaptosomes by a cyclic GMP-mediated mechanism. [J] . Mattson MP, Guo ZH, Geiger JD Journal of Neurochemistry: Offical Journal of the International Society for Neurochemistry . 1999,第2期

机译：淀粉样蛋白前体蛋白的分泌形式通过循环GMP介导的机制增强了突触小体中的基础葡萄糖和谷氨酸转运，并防止了葡萄糖和谷氨酸转运的氧化损伤。
2. Impairment of glucose and glutamate transport and induction of mitochondrial oxidative stress and dysfunction in synaptosomes by amyloid beta-peptide: role of the lipid peroxidation product 4-hydroxynonenal. [J] . Keller JN, Pang Z, Geddes JW, Journal of Neurochemistry: Offical Journal of the International Society for Neurochemistry . 1997,第1期

机译：淀粉样β肽对葡萄糖和谷氨酸转运的损害以及在突触小体中诱导线粒体氧化应激和功能障碍：脂质过氧化产物4-hydroxynonenal的作用。
3. Neurotrophic factors protect cortical synaptic terminals against amyloid and oxidative stress-induced impairment of glucose transport, glutamate transport and mitochondrial function. [J] . Guo ZH, Mattson MP Cerebral cortex . 2000,第1期

机译：神经营养因子保护皮质突触末端免受淀粉样蛋白和氧化应激诱导的葡萄糖转运，谷氨酸转运和线粒体功能的损害。
4. Growth Mechanism vs Matter Transport in Thermally Growing Oxides on High Temperature Materials: A Brief Survey Based on the Case Study of Alumina Formers [C] . J. Jedlinski International conference on diffusion in materials . 2009

机译：高温材料在热生长氧化物中的生长机制与氧化铝造粒机案例研究的简要调查
5. Modified electrodes for amperometric determination of glucose and glutamate using mediated electron transport. [D] . Harper, Alice C. 2005

机译：修饰的电极，用于通过介导的电子传输安培测定葡萄糖和谷氨酸。
6. Conditional Deletion of the Glutamate Transporter GLT-1 Reveals That Astrocytic GLT-1 Protects against Fatal Epilepsy While Neuronal GLT-1 Contributes Significantly to Glutamate Uptake into Synaptosomes [O] . Geraldine T. Petr, Yan Sun, Natalie M. Frederick, 2015

机译：谷氨酸转运蛋白GLT-1的条件性删除表明星形胶质细胞GLT-1保护免受致命性癫痫而神经元GLT-1明显促进谷氨酸摄入突触体中。
7. Impairment of Glucose and Glutamate Transport and Induction of Mitochondrial Oxidative Stress and Dysfunction in Synaptosomes by Amyloid β-Peptide: Role of the Lipid Peroxidation Product 4-Hydroxynonenal [O] . Jeffrey N. Keller, Zheng Pang, James W. Geddes, 2002

机译：糖苷β-肽抑制葡萄糖和谷氨酸输送和诱导线粒体氧化应激和功能障碍的诱导：脂质过氧化产物4-羟基的作用
8. Molecular mechanisms of enhanced [18F] fluorodeoxy glucose (FDG) uptake in isochemically injured myocardium: the role of glucose transporter and hexokinase expression. Final technical report for period August 1, 1993--November 30, 1997 [R] . 1999

机译：在化学损伤的心肌中增强的[18 F]氟脱氧葡萄糖（FDG）摄取的分子机制：葡萄糖转运蛋白和己糖激酶表达的作用。 1993年8月1日至1997年11月30日期间的最终技术报告

Secreted Form of Amyloid Precursor Protein Enhances Basal Glucose and Glutamate Transport and Protects Against Oxidative Impairment of Glucose and Glutamate Transport in Synaptosomes by a Cyclic GMP-Mediated Mechanism

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