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Prostate Cancer Cells Express More Androgen Receptor (AR) Following Androgen Deprivation, Improving Recognition by AR-Specific T Cells

机译：前列腺癌细胞表达了雄激素剥夺后的雄激素受体（AR），通过AR特异性T细胞改善识别

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Brian M. Olson; Melissa Gamat; Joseph Seliski; Thomas Sawicki; Justin Jeffery; Leigh Ellis; Charles G. Drake; Jamey Weichert; Douglas G. McNeel;
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1. Prostate Cancer Cells Express More Androgen Receptor (AR) Following Androgen Deprivation, Improving Recognition by AR-Specific T Cells [J] . Brian M. Olson, Melissa Gamat, Joseph Seliski, Cancer immunity . 2017,第12期

机译：前列腺癌细胞在雄激素被剥夺后表达更多的雄激素受体（AR），从而增强了AR特异性T细胞的识别能力
2. Targeted disruption of the p160 coactivator interface of androgen receptor (AR) selectively inhibits AR activity in both androgen-dependent and castration-resistant AR-expressing prostate cancer cells [J] . NakkaM., AgoulnikI.U., WeigelN.L. The international journal of biochemistry and cell biology . 2013,第4期

机译：靶向破坏雄激素受体（AR）的p160共激活因子接口选择性抑制雄激素依赖性和去势抵抗的AR表达前列腺癌细胞中的AR活性
3. Melatonin Inhibits Androgen Receptor Splice Variant-7 (AR-V7)-Induced Nuclear Factor-Kappa B (NF-κB) Activation and NF-κB Activator-Induced AR-V7 Expression in Prostate Cancer Cells: Potential Implications for the Use of Melatonin in Castration-Resistant Prostate Cancer (CRPC) Therapy [J] . Vincent Wing Sun Liu, Wing Lung Yau, Chun Wai Tam, International Journal of Molecular Sciences . 2017,第6期

机译：褪黑素抑制前列腺癌细胞中雄激素受体剪接变异体7（AR-V7）诱导的核因子-κB（NF-κB）活化和NF-κB活化剂诱导的AR-V7表达：在体内使用褪黑激素的潜在影响去势抵抗性前列腺癌（CRPC）治疗
4. Role of the Androgen Receptor and PI3K/Akt in the Survival of Androgen-Refractory Prostate Cancer Cells [C] . Haojie Huang, Donald J. Tindal International Symposium on Hormonal Carcinogenesis . 2005

机译：雄激素受体和PI3K / AKT在雄激素难治性前列腺癌细胞存活中的作用
5. MED19 Alters Genomic Occupancy of the Androgen Receptor and Gene Expression in Prostate Cancer Cells, Driving Growth under Low Androgen [D] . Weber, Hannah. 2020

机译：Med19改变了前列腺癌细胞中雄激素受体和基因表达的基因组占据，在低雄激素下促进生长
6. Prostate cancer cells express more androgen receptor (AR) following androgen deprivation improving recognition by AR-specific T cells [O] . Brian M. Olson, Melissa Gamat, Joseph Seliski, -1

机译：前列腺癌细胞在雄激素被剥夺后表达更多的雄激素受体（AR）从而提高了AR特异性T细胞的识别能力
7. Targeted disruption of the p160 coactivator interface of androgen receptor (AR) selectively inhibits AR activity in both androgen-dependent and castration-resistant AR-expressing prostate cancer cells [O] . Manjula Nakka, Irina U. Agoulnik, Nancy L. Weigel 2013

机译：雄激素受体（Ar）的P160共粘膜界面的靶向破坏选择性地抑制雄激素依赖性和抗阉割的抗溶胀AR的前列腺癌细胞中的AR活性
8. Induced Expression of Androgen Receptor in Androgen Independent Prostate Cancer Cells Using an I kBa 'Super Repressor' [R] . Kalikin, L. M. , Pienta, K. J. 2004

机译：使用I kBa'超级抑制因子诱导雄激素受体在雄激素非依赖性前列腺癌细胞中的表达

Prostate Cancer Cells Express More Androgen Receptor (AR) Following Androgen Deprivation, Improving Recognition by AR-Specific T Cells

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