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Morphology, immunophenotype, and distribution of latently and/or productively Epstein-Barr virus-infected cells in acute infectious mononucleosis: implications for the interindividual infection route of Epstein-Barr virus

机译：急性传染性单核细胞增多症中潜伏期，免疫蛋白型和潜伏的epstein-BARR病毒感染细胞的形态学，免疫蛋白型和分布：对Epstein-Barr病毒的联系感染途径的影响

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I Anagnostopoulos; M Hummel; C Kreschel; H Stein;
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1. Epstein-Barr virus (EBV) infection in infectious mononucleosis: virus latency, replication and phenotype of EBV-infected cells. [J] . Niedobitek G, Agathanggelou A, Herbst H, Journal of Pathology: Journal of the Pathological Society of Great Britain and Ireland . 1997,第2期

机译：传染性单核细胞增多症中的爱泼斯坦-巴尔病毒（EBV）感染：EBV感染细胞的病毒潜伏期，复制和表型。
2. Cytokine-Mediated Loss of Blood Dendritic Cells During Epstein-Barr Virus-Associated Acute Infectious Mononucleosis: Implication for Immune Dysregulation [J] . Panikkar Archana, Smith Corey, Hislop Andrew, The Journal of Infectious Diseases . 2015,第12期

机译：在爱泼斯坦-巴尔病毒相关的急性感染性单核细胞增多症期间细胞因子介导的树突状细胞损失：免疫失调的影响。
3. Plasmacytic differentiation of circulating Epstein-Barr virus-infected B lymphocytes during acute infectious mononucleosis. [J] . J E Robinson, D Smith, J Niederman The Journal of Experomental Medicine . 1981,第2期

机译：急性感染性单核细胞增多症过程中循环爱泼斯坦-巴尔病毒感染的B淋巴细胞的血浆细胞分化。
4. Virus-Host Interactions during Epstein-Barr virus Latent Infection of B cells [D] . Homa, Nicholas Joseph. 2017

机译：B细胞的爱泼斯坦-巴尔病毒潜伏感染期间的病毒-宿主相互作用
5. The Switch from Latent to Productive Infection in Epstein-Barr Virus-Infected B Cells Is Associated with Sensitization to NK Cell Killing [O] . Isabel Y. Pappworth, Eddie C. Wang, Martin Rowe 2007

机译：在爱泼斯坦-巴尔病毒感染的B细胞中从潜伏感染向生产性感染的转变与致敏性相关联从而杀伤NK细胞。
6. The Switch from Latent to Productive Infection in Epstein-Barr Virus-Infected B Cells Is Associated with Sensitization to NK Cell Killing▿ [O] . Pappworth, Isabel Y., Wang, Eddie C., Rowe, Martin 2006

机译：爱泼斯坦-巴尔病毒感染的B细胞从潜伏感染向生产性感染的转变与致敏性杀伤NK细胞有关▿

Morphology, immunophenotype, and distribution of latently and/or productively Epstein-Barr virus-infected cells in acute infectious mononucleosis: implications for the interindividual infection route of Epstein-Barr virus

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