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Lack of Effective MUC1 Tumor Antigen-Specific Immunity in MUC1-Transgenic Mice Results from a Th/T Regulatory Cell Imbalance That Can Be Corrected by Adoptive Transfer of Wild-Type Th Cells

机译：在Muc1-转基因小鼠中缺乏有效的Muc1肿瘤抗原特异性免疫来自TH / T调节细胞不平衡，这可以通过野生型细胞的通过转移来校正

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Michael S. Turner; Patrice A. Cohen; Olivera J. Finn;
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1. Lack of Effective MUC1 Tumor Antigen-Specific Immunity in MUC1-Transgenic Mice Results from a Th/T Regulatory Cell Imbalance That Can Be Corrected by Adoptive Transfer of Wild-Type Th Cells [J] . Michael S. Turner, Patrice A. Cohen, Olivera J. Finn The journal of immunology . 2007,第5期

机译：Th / T调节性细胞失衡可导致MUC1转基因小鼠缺乏有效的MUC1肿瘤抗原特异性免疫，可以通过野生型Th细胞的过继转移来纠正
2. NK cell adoptive transfer combined with ontak-mediated regulatory T cell elimination induces effective adaptive antitumor immune responses. [J] . Salagianni M, Lekka E, Moustaki A, The Journal of Immunology: Official Journal of the American Association of Immunologists . 2011,第6期

机译：NK细胞过继转移结合ontak介导的调节性T细胞消除诱导有效的适应性抗肿瘤免疫反应。
3. NK Cell Adoptive Transfer Combined with Ontak-Mediated Regulatory T Cell Elimination Induces Effective Adaptive Antitumor Immune Responses [J] . Maria Salagianni, Eftychia Lekka, Ardiana Moustaki, The journal of immunology . 2011,第6期

机译：NK细胞过继转移结合Ontak介导的调节性T细胞消除诱导有效的自适应抗肿瘤免疫反应。
4. Modulation of tumor response to photodynamic therapy in severe combined immunodeficient (SCID) mice by adoptively transferred lymphoid cells [C] . Mladen Korbelik, British Columbia Cancer Agency, Vancouver, Optical Methods for Tumor Treatment and Detection: Mechanisms and Techniques in Photodynamic Therapy V . 1996

机译：过继转移淋巴样细胞对重度联合免疫缺陷（SCID）小鼠对光动力疗法的肿瘤反应的调节
5. Endogenous antitumor immunity elicited by adoptively transferred lymphocytes through CCL5 and CD40L signaling, and dendritic cell depletion. [D] . Nesbeth, Yolanda C. 2010

机译：过继转移的淋巴细胞通过CCL5和CD40L信号传导以及树突状细胞耗竭引发内源性抗肿瘤免疫力。
6. Nonatopic asthma: in vivo airway hyperreactivity adoptively transferred to naive mice by THY-1(+) and B220(+) antigen-specific cells that lack surface expression of CD3. [O] . G P Geba, C D Wegner, W W Wolyniec, 1997

机译：非特应性哮喘：缺乏CD3表面表达的THY-1（+）和B220（+）抗原特异性细胞将体内气道高反应性过继转移至幼稚小鼠。
7. Three Different Vaccines Based on the 140-Amino Acid MUC1 Peptide with Seven Tandemly Repeated Tumor-Specific Epitopes Elicit Distinct Immune Effector Mechanisms in Wild-Type Versus MUC1-Transgenic Mice with Different Potential for Tumor Rejection [O] . M. Melina Soares, Vinay Mehta, Olivera J. Finn 2001

机译：基于140-氨基酸MUC1肽的三种不同的疫苗，具有七个串联重复的肿瘤特异性表位引发了野生型免疫效应机制的不同肿瘤排斥潜力的不同潜力
8. Requirement for Continuous Antigenic Stimulation of Immune Spleen Cells in Adoptive Transfer of Cell-Mediated Protection against Tularemia. [R] . Hunter, D. H., Eigelsbach, H. T., McGann, V. G., 1976

机译：在过继转移细胞介导的Tularemia保护中持续抗原刺激免疫脾细胞的要求。

Lack of Effective MUC1 Tumor Antigen-Specific Immunity in MUC1-Transgenic Mice Results from a Th/T Regulatory Cell Imbalance That Can Be Corrected by Adoptive Transfer of Wild-Type Th Cells

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