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Continuous and intermittent exposure of neonatal rat calvarial cells to PTHrP (1-36) inhibits bone nodule mineralization in vitro by downregulating bone sialoprotein expression via the cAMP signaling pathway

机译：通过CAMP信号通路下调骨唾液蛋白表达，对新生大鼠颅骨细胞的连续和间歇暴露于PTHRP（1-36）抑制骨结节矿化

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Suzan A Kamel; John A Yee;
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1. Biosynthesis of bone proteins [SPP-1 (secreted phosphoprotein-1, osteopontin), BSP (bone sialoprotein) and SPARC (osteonectin)] in association with mineralized-tissue formation by fetal-rat calvarial cells in culture [J] . C G Bellows, J Sodek, S Kasugai, The biochemical journal . 1991,第2期

机译：骨蛋白[SPP-1（分泌的磷蛋白-1，骨桥蛋白），BSP（骨唾液蛋白）和SPARC（骨连接蛋白）的生物合成）与培养的胎鼠颅盖骨细胞形成矿化组织有关
2. Intermittent parathyroid hormone (1-34) application regulates cAMP-response element binding protein activity to promote the proliferation and osteogenic differentiation of bone mesenchymal stromal cells, via the cAMP/PKA signaling pathway [J] . Chen Bailing, Lin Tao, Yang Xiaoxi, Experimental and therapeutic medicine . 2016,第6期

机译：间歇性甲状旁腺激素（1-34）的应用通过cAMP / PKA信号通路调节cAMP反应元件结合蛋白的活性，从而促进骨间充质基质细胞的增殖和成骨分化
3. Formation of mineralized bone nodules by rat calvarial osteoblasts decreases with donor age due to a reduction in signaling through EP(1) subtype of prostaglandin E(2) receptor. [J] . Fujieda M, Kiriu M, Mizuochi S, Journal of cellular biochemistry. . 1999,第2期

机译：大鼠颅盖成骨细胞形成矿化骨结节的形成随着供体年龄的增加而减少，这是由于通过前列腺素E（2）受体的EP（1）亚型减少了信号传导。
4. Exogenous Runx2/Cbfa1 Expression in Bone Marrow Stromal Cells Enhances Matrix Mineralization of 3-D Polymeric Scaffolds In Vitro and In Vivo [C] . Benjamin A. Byers, Robert E. Guldberg, Andrés J. García Annual meeting of the Society For Biomaterials . 2003

机译：骨髓基质细胞中外源Runx2 / Cbfa1表达增强了3D聚合物支架体外和体内的基质矿化作用。
5. Portulaca Oleracea Extract Can Inhibit Nodule Formation of Colon Cancer Stem Cells by Regulating Gene Expression of Notch Signal Transduction Pathway [D] . Chen, Li. 2016

机译：马齿ula提取物可通过调节Notch信号转导通路的基因表达抑制结肠癌干细胞的结节形成
6. Continuous and intermittent exposure of neonatal rat calvarial cells to PTHrP (1-36) inhibits bone nodule mineralization in vitro by downregulating bone sialoprotein expression via the cAMP signaling pathway [O] . Suzan A Kamel, John A Yee, Joseph Bidwell, -1

机译：新生大鼠颅盖细胞连续和间歇性暴露于PTHrP（1-36）可通过下调cAMP信号通路的骨唾液蛋白表达来抑制体外的骨结节矿化
7. Continuous and intermittent exposure of neonatal rat calvarial cells to PTHrP (1-36) inhibits bone nodule mineralization in vitro by downregulating bone sialoprotein expression via the cAMP signaling pathway [O] . Suzan A Kamel, John A Yee 2013

机译：通过CAMP信号通路下调骨唾液蛋白表达，对新生大鼠颅骨细胞的连续和间歇暴露于PTHRP（1-36）抑制骨结节矿化

Continuous and intermittent exposure of neonatal rat calvarial cells to PTHrP (1-36) inhibits bone nodule mineralization in vitro by downregulating bone sialoprotein expression via the cAMP signaling pathway

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