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Novel therapeutic strategy for advanced prostate cancer using antisense oligodeoxynucleotides targeting anti-apoptotic genes upregulated after androgen withdrawal to delay androgen-independent progression and enhance chemosensitivity

机译：使用反义寡脱氧核苷酸的新型前列腺癌的新型治疗策略靶向抗凋亡基因在雄激素戒断后上调，延迟雄激素无关的进展，增强化学敏感性

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Hideaki Miyake; Isao Hara; Sadao Kamidono; Martin E Gleave;
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年度 2001
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1. Targeting Anti-Apoptotic Genes Upregulated by Androgen Withdrawal Using Antisense Oligonucleotides to Enhance Androgen- and Chemo-Sensitivity in Prostate Cancer [J] . Martin E. Gleave, Toby Zellweger, Kim Chi, Investigational New Drugs . 2002,第2期

机译：通过使用反义寡核苷酸增强雄激素戒断的上调抗凋亡基因，以增强前列腺癌的雄激素和化学敏感性
2. Targeting anti-apoptotic genes upregulated by androgen withdrawal using antisense oligonucleotides to enhance androgen- and chemo-sensitivity in prostate cancer. [J] . Gleave ME, Zellweger T, Chi K, Investigational new drugs. . 2002,第2期

机译：使用反义寡核苷酸通过雄激素戒断上调靶向抗凋亡基因，以增强前列腺癌中的雄激素敏感性和化学敏感性。
3. Chemosensitization and delayed androgen-independent recurrence of prostate cancer with the use of antisense Bcl-2 oligodeoxynucleotides. [J] . Miayake H, Tolcher A, Gleave ME Journal of the National Cancer Institute . 2000,第1期

机译：使用反义Bcl-2寡脱氧核苷酸可引起前列腺癌的化学增敏作用和延迟的雄激素非依赖性复发。
4. Signal transduction targets in androgen-independent prostate cancer [C] . Jian Zhou, Jessica Scholes, Jer-Tsong Hsieh World Congress on Heart Failure--Mechanisms and Management . 2002

机译：信号转导靶向雄激素无关的前列腺癌
5. Effect of androgen and natural compounds on proliferation of androgen-independent LNCaP human prostate cancer cells and prostate cancer progression. [D] . Chuu, Chih-pin. 2005

机译：雄激素和天然化合物对雄激素非依赖性LNCaP人前列腺癌细胞增殖和前列腺癌进展的影响。
6. Therapeutic Efficacy of Adenoviral-Mediated p53 Gene Transfer Is Synergistically Enhanced by Combined Use of Antisense Oligodeoxynucleotide Targeting Clusterin Gene in a Human Bladder Cancer Model [O] . Hideaki Miyake, Kazuki Yamanaka, Mototsugu Muramaki, 2005

机译：腺病毒介导的p53基因转移的治疗功效通过在人膀胱癌模型中联合使用靶向Clusterin基因的反义寡脱氧核苷酸而得到协同增强
7. A novel antisense oligonucleotide inhibiting several antiapoptotic Bcl-2 family members induces apoptosis and enhances chemosensitivity in androgen-independent human prostate cancer PC3 cells [O] . Kazuki Yamanaka, Palma Rocchi, Hideaki Miyake, 2005

机译：一种抑制几种抗透露的Bcl-2家族成员的新型反义寡核苷酸诱导细胞凋亡并增强雄激素无关的人前列腺癌PC3细胞的化学敏感性
8. Molecular Determinants of Prostate Cancer Progression Across Race- Ethnicity. Project A - The Human 5RD5A2 Gene and Prostate Cancer Progression. Project B - Androgen Receptor (AR) Signaling in Prostate Cancer Progression. Project C - Cellular and Molecular Markers of Prostate Cancer Progression Core - Epidemiology Core [R] . Ross, R. R. , Reichardt, J. , Coetzee, G. A. , 2002

机译：跨越种族的前列腺癌进展的分子决定因素。项目a - 人类5RD5a2基因和前列腺癌进展。项目B - 雄激素受体（aR）在前列腺癌进展中的信号传导。项目C - 前列腺癌进展核心的细胞和分子标记 - 流行病学核心

Novel therapeutic strategy for advanced prostate cancer using antisense oligodeoxynucleotides targeting anti-apoptotic genes upregulated after androgen withdrawal to delay androgen-independent progression and enhance chemosensitivity

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