首页> 外文OA文献 >Loss of the Protein NUPR1 (p8) Leads to Delayed LHB Expression, Delayed Ovarian Maturation, and Testicular Development of a Sertoli-Cell-Only Syndrome-Like Phenotype in Mice1

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Loss of the Protein NUPR1 (p8) Leads to Delayed LHB Expression, Delayed Ovarian Maturation, and Testicular Development of a Sertoli-Cell-Only Syndrome-Like Phenotype in Mice1

机译：蛋白质NUPR1（p8）的丧失导致LHB表达延迟，延迟卵巢成熟，并且在小鼠中睾丸-MICS-ock-ockidOm样表型的睾丸发育。

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Christina M. Million Passe; Crystal R. White; Michael W. King; Phillip L. Quirk; Juan L. Iovanna; Christine C. Quirk;
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年度 2008
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1. Loss of the protein NUPR1 (p8) leads to delayed LHB expression, delayed ovarian maturation, and testicular development of a sertoli-cell-only syndrome-like phenotype in mice. [J] . Million-Passe CM, White CR, King MW, Biology of Reproduction: Offical Journal of the Society for the Study of Reproduction . 2008,第4期

机译：蛋白质NUPR1（p8）的缺失会导致LHB表达延迟，卵巢成熟延迟以及小鼠睾丸仅睾丸综合征综合征表型的睾丸发育。
2. Fragile X astrocytes induce developmental delays in dendrite maturation and synaptic protein expression [J] . Shelley Jacobs, Meera Nathwani, Laurie C Doering BMC Neuroscience . 2010,第1期

机译：脆弱的X星形胶质细胞诱导树突成熟和突触蛋白表达的发育延迟
3. Inhibition of Skp1-Cullin-F-box complexes during bovine oocyte maturation and preimplantation development leads to delayed development of embryos [J] . Kinterova Veronika, Kanka Jiri, Petruskova Veronika, Biology of Reproduction: Offical Journal of the Society for the Study of Reproduction . 2019,第4期

机译：在牛卵母细胞成熟期间的SKP1-Cullin-F盒复合物的抑制导致胚胎的延迟发育
4. Fragile X astrocytes induce developmental delays in dendrite maturation and synaptic protein expression [O] . Shelley Jacobs, Meera Nathwani, Laurie C Doering 2010

机译：易碎的X星形胶质细胞诱导树突成熟和突触蛋白表达的发育延迟
5. Fragile X astrocytes induce developmental delays in dendrite maturation and synaptic protein expression [O] . Doering Laurie C, Nathwani Meera, Jacobs Shelley 2010

机译：脆弱的X星形胶质细胞诱导树突成熟和突触蛋白表达的发育延迟

Loss of the Protein NUPR1 (p8) Leads to Delayed LHB Expression, Delayed Ovarian Maturation, and Testicular Development of a Sertoli-Cell-Only Syndrome-Like Phenotype in Mice1

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