首页> 外文OA文献 >RANTES (CCL5) induces a CCR5-dependent accelerated shedding of syndecan-1 (CD138) and syndecan-4 from HeLa cells and forms complexes with the shed ectodomains of these proteoglycans as well as with those of CD44

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RANTES (CCL5) induces a CCR5-dependent accelerated shedding of syndecan-1 (CD138) and syndecan-4 from HeLa cells and forms complexes with the shed ectodomains of these proteoglycans as well as with those of CD44

机译：Rantes（CCL5）从HeLa细胞中诱导CCR5依赖性加速脱落的Syndecan-1（CD138）和Syndecan-4，并与这些蛋白多糖的血迹和CD44的抗斑块形成复合物

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N. Charnaux;
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1. RANTES (CCL5) induces a CCR5-dependent accelerated shedding of syndecan-1 (CD138) and syndecan-4 from HeLa cells and forms complexes with the shed ectodomains of these proteoglycans as well as with those of CD44. [J] . Charnaux N, Brule S, Chaigneau T, Glycobiology. . 2005,第2期

机译：RANTES（CCL5）诱导HeLa细胞上syndecan-1（CD138）和syndecan-4的CCR5依赖性加速脱落，并与这些蛋白聚糖的脱落胞外域以及CD44的胞外域形成复合体。
2. The shedding of syndecan-4 and syndecan-1 from HeLa cells and human primary macrophages is accelerated by SDF-1/CXCL12 and mediated by the matrix metalloproteinase-9. [J] . Brule S, Charnaux N, Sutton A, Glycobiology. . 2006,第6期

机译：SDF-1 / CXCL12加速了HeLa细胞和人类原代巨噬细胞中syndecan-4和syndecan-1的脱落，并由基质金属蛋白酶9介导。
3. The shedding of syndecan-4 and syndecan-1 from HeLa cells and human primary macrophages is accelerated by SDF-1/CXCL12 and mediated by the matrix metalloproteinase-9 [J] . Severine Brule, Nathalie Charnaux, Angela Sutton, Glycobiology . 2006,第6期

机译：SDF-1 / CXCL12加速HeLa细胞和人类原代巨噬细胞中syndecan-4和syndecan-1的脱落，并由基质金属蛋白酶9介导
4. Microscopy Techniques and Technologies Used in Ultrastructural Analyses of Poliovirus Replication Complexes Formed During Infection of HeLa Cells [C] . ER Fischer, V Nair, BT Hansen, Microscopy and microanalysis.;Microscopy and microanalysis. . -1

机译：显微镜技术和技术用于HeLa细胞感染过程中形成的脊髓灰质炎病毒复制复合物的超微结构分析
5. Heparanase-induced shedding of syndecan-1/CD138 in myeloma and endothelial cells activates VEGFR2 and an invasive phenotype: prevention by novel synstatins [O] . O Jung, V Trapp-Stamborski, A Purushothaman, 2016

机译：乙酰肝素酶诱导的骨髓瘤和内皮细胞中syndecan-1 / CD138的脱落激活了VEGFR2和一种侵袭性表型：通过新型synstatins进行预防
6. Binding of the CC-chemokine RANTES to syndecan-1 and syndecan-4 expressed on HeLa cells [O] . Hocine Slimani, Nathalie Charnaux, Line Saffar, 2003

机译：CC-趋化因子RaNTEs与HeLa细胞上表达的syndecan-1和syndecan-4的结合
7. Late Mitosis/Early G sub 1 Phase and Mid-G sub 1 Phase Are Not HypersensitiveCell Cycle Phases for Neoplastic Transformation of HeLa x Skin Fibroblast Human Hybrid Cells Induced by Fission-Spectrum Neutrons [R] . Redpath, J. L., Antoniono, R. J., Sun, C., 1995

机译：晚期有丝分裂/早期G 1期和中期G 1期不是过敏性细胞周期阶段用于裂变谱中子诱导的HeLa x皮肤成纤维细胞人类杂交细胞的肿瘤转化

RANTES (CCL5) induces a CCR5-dependent accelerated shedding of syndecan-1 (CD138) and syndecan-4 from HeLa cells and forms complexes with the shed ectodomains of these proteoglycans as well as with those of CD44

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