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The Tyr-Kinase Inhibitor AG879, that Blocks the ETK-PAK1 Interaction, Suppresses the RAS-induced PAK1 Activation and Malignant Transformation

机译：阻断ETK-PAK1相互作用的Tyr-激酶抑制剂AG879抑制了RAS诱导的PAK1活化和恶性转化

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Hong He; Yumiko Hirokawa; Aviv Gazit; Yoshihiro Yamashita; Hiroyuki Mano; Yuko Kawakami; Toshiaki Kawakami; Ching-Yi Hsieh; Hsing-Jien Kung; Guillaume Lessene; Jonathan Baell; Alexander Levitzki; Hiroshi Maruta;
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年度 2004
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1. The Tyr-kinase inhibitor AG879, that blocks the ETK-PAK1 interaction, suppresses the RAS-induced PAK1 activation and malignant transformation. [J] . He H, Hirokawa Y, Gazit A, Cancer biology & therapy . 2004,第1期

机译：阻止ETK-PAK1相互作用的Tyr激酶抑制剂AG879抑制RAS诱导的PAK1活化和恶性转化。
2. The Tyr-kinase inhibitor AG879, that blocks the ETK-PAK1 interaction, suppresses the RAS-induced PAK1 activation and malignant transformation. [J] . He H, Hirokawa Y, Gazit A, Cancer biology & therapy . 2004,第1期

机译：阻止ETK-PAK1相互作用的Tyr激酶抑制剂AG879抑制RAS诱导的PAK1活化和恶性转化。
3. BET Bromodomain Suppression Inhibits VEGF-induced Angiogenesis and Vascular Permeability by Blocking VEGFR2-mediated Activation of PAK1 and eNOS [J] . Mingcheng Huang, Qian Qiu, Youjun Xiao, Scientific reports. . 2016,第1期

机译：BET溴结构域抑制通过阻断VEGFR2介导的PAK1和eNOS的激活来抑制VEGF诱导的血管生成和血管通透性。
4. AcSDKP inhibits the proliferation and collagen expression of cardiac fibroblasts induced by PDGF through blocking the ERK1/2 and JNK pathway activation [C] . Zhang Lijuan, Yang Fang, Li Qian, 2011 International Conference on Human Health and Biomedical Engineering . 2011

机译：AcSDKP通过阻断ERK1 / 2和JNK途径的激活来抑制PDGF诱导的心脏成纤维细胞的增殖和胶原蛋白表达
5. Part I. Solvolysis regioselectivity and x-ray structure of the duocarmycin SA alkylation subunit. Part II. Solution phase synthesis of compound libraries targeting the erythropoietin receptor. Part III. Identification of a new class of angiogenesis inhibitors which function by blocking the interaction of proteinase MMP2 and integrin alpha(v)beta(3) [D] . Goldberg, Joel Adam 2000

机译：第一部分：杜卡霉素SA烷基化亚基的溶剂分解区域选择性和X射线结构。第二部分靶向促红细胞生成素受体的化合物库的溶液相合成。第三部分通过阻断蛋白酶MMP2和整联蛋白α（v）beta（3）的相互作用来识别新型血管生成抑制剂
6. BET Bromodomain Suppression Inhibits VEGF-induced Angiogenesis and Vascular Permeability by Blocking VEGFR2-mediated Activation of PAK1 and eNOS [O] . Mingcheng Huang, Qian Qiu, Youjun Xiao, -1

机译：BET溴结构域抑制通过阻断VEGFR2介导的PAK1和eNOS的激活来抑制VEGF诱导的血管生成和血管通透性。
7. BET Bromodomain Suppression Inhibits VEGF-induced Angiogenesis and Vascular Permeability by Blocking VEGFR2-mediated Activation of PAK1 and eNOS [O] . Mingcheng Huang, Qian Qiu, Youjun Xiao, 2016

机译：Bet Bromodomain抑制通过阻断VEGFR2介导的PAK1和ENOS的活化来抑制VEGF诱导的血管生成和血管渗透性
8. Enzyme MnSOD Suppresses Malignant Breast Cell Growth by Preventing HIF-1 Activation [R] . Wang, M. , Buettner, G. 2005

机译：酶mnsOD通过防止HIF-1活化来抑制恶性乳腺细胞生长

The Tyr-Kinase Inhibitor AG879, that Blocks the ETK-PAK1 Interaction, Suppresses the RAS-induced PAK1 Activation and Malignant Transformation

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