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Caffeine reduces deficits in mechanosensation and locomotion induced by L-DOPA and protects dopaminergic neurons in a transgenic Caenorhabditis elegans model of Parkinson’s disease

机译：咖啡因减少了L-DOPA诱导的机械化和运动的缺陷，并保护多巴胺能神经元在帕金森病的转基因Caenorhabdisegans模型中保护多巴胺能神经元

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Rafael Vincent M. Manalo; Paul Mark B. Medina;
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1. Caffeine reduces deficits in mechanosensation and locomotion induced by L -DOPA and protects dopaminergic neurons in a transgenic Caenorhabditis elegans model of Parkinson’s disease [J] . Rafael Vincent M. Manalo, Paul Mark B. Medina Pharmaceutical Biology . 2020,第1期

机译：咖啡因减少了L -Dopa诱导的机械淤积和运动的缺陷，并保护多巴胺能神经元在帕金森病的转基因Caenorhabdise秀丽叶子中
2. Caffeine Protects Dopaminergic Neurons From Dopamine-Induced Neurodegeneration via Synergistic Adenosine-Dopamine D2-Like Receptor Interactions in Transgenic Caenorhabditis elegans [J] . Rafael V. M. Manalo, Paul M. B. Medina Frontiers in Neuroscience . 2018,第2016期

机译：咖啡因通过协同腺苷-多巴胺D2-受体相互作用在转基因秀丽隐杆线虫中保护多巴胺能神经元免受多巴胺诱导的神经变性。
3. MANF protects dopamine neurons and locomotion defects from a human α-synuclein induced Parkinson's disease model in C. elegans by regulating ER stress and autophagy pathways [J] . Zhuoyu Zhang, Yijue Shen, Hang Luo, Experimental Neurology . 2018,第期

机译：通过调节ER压力和自噬途径，曼夫保护多巴胺神经元和人类α-突触核蛋白诱导的帕金森病模型的帕金森病模型。
4. Cerium Oxide Nanoparticles Protect Against MPTP-Induced Dopaminergic Neurodegeneration In A Mouse Model For Parkinson's Disease [C] . Dillon CE, Billings M, Hockey KS, NSTI nanotechnology conference and expo;Nanotech conference expo;NSTI-Nanotech 2011;TechConnect world annual conference expo . 2011

机译：氧化铈纳米颗粒可预防MPTP诱发的帕金森氏病小鼠模型中的多巴胺能神经变性
5. Molecular mechanisms of PKCdelta in neurotoxin-induced apoptotic death of nigral dopaminergic neurons: Relevance to the pathogenesis of Parkinson's disease. [D] . Yang, Yongjie. 2005

机译：神经毒素诱导的多巴胺能神经元凋亡死亡中PKCdelta的分子机制：与帕金森氏病的发病机制有关。
6. Caffeine reduces deficits in mechanosensation and locomotion induced by L-DOPA and protects dopaminergic neurons in a transgenic Caenorhabditis elegans model of Parkinson’s disease [O] . Rafael Vincent M. Manalo, Paul Mark B. Medina 2020

机译：咖啡因减少了L-DOPA诱导的机械化和运动的缺陷并保护多巴胺能神经元在帕金森病的转基因Caenorhabdisegans模型中保护多巴胺能神经元
7. n-butylidenephthalide protects against dopaminergic neuron degeneration and α-synuclein accumulation in Caenorhabditis elegans models of Parkinson's disease. [O] . Ru-Huei Fu, Horng-Jyh Harn, Shih-Ping Liu, 2014

机译：正丁基苯酞在帕金森氏病的秀丽隐杆线虫模型中防止多巴胺能神经元变性和α-突触核蛋白积聚。

Caffeine reduces deficits in mechanosensation and locomotion induced by L-DOPA and protects dopaminergic neurons in a transgenic Caenorhabditis elegans model of Parkinson’s disease

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