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CRISPR/Cas9-Based Knockout of GNAQ Reveals Differences in Host Cell Signaling Necessary for Egress of Apicomplexan Parasites

机译：基于CRISPR / CAS9的GNAQ敲除揭示了APICOMPLAMAN寄生虫出口所需的宿主细胞信号的差异

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Paul-Christian Burda; Hugo Bisio; Jean-Baptiste Marq; Dominique Soldati-Favre; Volker T. Heussler;
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1. CRISPR/Cas9-Based Knockout of GNAQ Reveals Differences in Host Cell Signaling Necessary for Egress of Apicomplexan Parasites [J] . Paul-Christian Burda, Hugo Bisio, Jean-Baptiste Marq, mSphere . 2020,第6期

机译：基于CRISPR / CAS9的GNAQ敲除揭示了APICOMPLAMAN寄生虫出口所需的宿主细胞信号的差异
2. CRISPR/Cas9-based Knockout Strategy Elucidates Components Essential for Type 1 Interferon Signaling in Human HeLa Cells [J] . Urin Victoria, Shemesh Maya, Schreiber Gideon Journal of Molecular Biology . 2019,第17期

机译：基于CRISPR / CAS9的敲除策略阐明了人Hela细胞中1型干扰素信号所必需的组分
3. A host GPCR signaling network required for the cytolysis of infected cells facilitates release of apicomplexan parasites. [J] . Millholland M. G., Mishra S., Dupont C. D., Cell Host & Microbe . 2013,第1期

机译：感染细胞的细胞溶解所需的宿主GPCR信号网络可促进复合体寄生虫的释放。
4. Chemical profile differences in endemic parasitic weeds: a study of host-parasite chemical profiles in select mistletoe and eucalypt species [C] . Aaron L. Preston, Min An, David M. Watson Australian Weeds Conferences . 2010

机译：流行寄生杂草中的化学型材差异：选择槲寄生和桉树物种中寄生虫化学型材的研究
5. Diversity, infection patterns and host-parasite associations of apicomplexan parasites in reptiles [D] . Maia, Joao Pedro Moura da Costa 2015

机译：爬行动物中ApiCoMplexan寄生虫的多样性，感染模式和寄生虫寄生虫关联
6. CRISPR/Cas9-Based Knockout of GNAQ Reveals Differences in Host Cell Signaling Necessary for Egress of Apicomplexan Parasites [O] . Paul-Christian Burda, Hugo Bisio, Jean-Baptiste Marq, 2020

机译：基于CRISPR / CAS9的GNAQ敲除揭示了APICOMPLAMAN寄生虫出口所需的宿主细胞信号的差异
7. CRISPR/Cas9-based knockouts reveal that CpRLP1 is a negative regulator of the sex pheromone PR-IP in the Closterium peracerosum-strigosum-littorale complex [O] . Naho Kanda, Machiko Ichikawa, Ayaka Ono, 2017

机译：基于CRISPR / CAS9的敲除揭示了CPRLP1是Peracerosum-strigosum-Littorale复合物中的性信息素PR-IP的阴性调节器

CRISPR/Cas9-Based Knockout of GNAQ Reveals Differences in Host Cell Signaling Necessary for Egress of Apicomplexan Parasites

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