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DOP002 Conditional deletion of Prep1 in the intestinal epithelium alters epithelial homeostasis, intestinal development, and controls colitis susceptibility

机译：DOP002条件删除预备1在肠上皮内改变上皮稳态，肠道发育和控制结肠炎易感性

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S. DAlessio; C. Tacconi; A. Gandelli; C. Correale; M. Genua; S. Vetrano; F. Blasi; S. Danese;
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年度 2014
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1. Altered intestinal epithelial homeostasis in mice with intestine-specific deletion of the Kruppel-like factor 4 gene. [J] . Ghaleb AM, McConnell BB, Kaestner KH, Developmental biology . 2011,第2期

机译：肠道特异性Kruppel样因子4基因缺失的小鼠肠道上皮稳态改变。
2. Altered intestinal epithelial homeostasis in mice with intestine-specific deletion of the Krüppel-like factor 4 gene [J] . Amr M. Ghaleb, Beth B. McConnell, Klaus H. Kaestner, Developmental biology . 2011,第2期

机译：肠道特异性删除Kr＆amp; upel-like factor 4基因的小鼠肠道上皮稳态改变
3. A31 ALTERED SMALL INTESTINAL EPITHELIAL HOMEOSTASIS AND CELL FATE DECISION UPON LOSS OF TRP53 IN KRT15 INTESTINAL STEM CELLS [J] . Dubey A, Giroux V Journal of the Canadian Association of Gastroenterology . 2020,第Supplementa1期

机译：A31改变了小肠上皮稳态和细胞命运在KRT15肠道干细胞中丧失TRP53时的决定
4. Synthetic Hydrogel Scaffolds for Modeling Intestinal Epithelial Homeostasis In Vitro [C] . Reid L. Wilson, K. Jane Grande-Allen Society for Biomaterials annual meeting and exposition . 2017

机译：体外模拟肠道上皮稳态的合成水凝胶支架
5. Cholinergically-mediated intestinal epithelial ion transport in the dextran sodium sulphate model of colitis in mice. [D] . Hirota (ne Green), Christina L. 2007

机译：在小鼠结肠炎的葡聚糖硫酸钠模型中，胆碱介导的肠上皮离子运输。
6. Conditional deletion of β1 integrins in the intestinal epithelium causes a loss of Hedgehog expression intestinal hyperplasia and early postnatal lethality [O] . Robert G. Jones, Xiufen Li, Phillip D. Gray, 2006

机译：肠上皮细胞中β1整合素的条件缺失会导致刺猬蛋白表达下降肠增生和产后早期致死率
7. Altered intestinal epithelial homeostasis in mice with intestine-specific deletion of the Krüppel-like factor 4 gene [O] . Ghaleb Amr M., McConnell Beth B., Kaestner Klaus H., 2011

机译：肠道特异性Krüppel样因子4基因缺失的小鼠肠道上皮稳态改变

DOP002 Conditional deletion of Prep1 in the intestinal epithelium alters epithelial homeostasis, intestinal development, and controls colitis susceptibility

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