首页> 外文OA文献 >Prx1 Suppresses Radiation-Induced c-Jun NH2-Terminal Kinase Signaling in Lung Cancer Cells through Interaction with the Glutathione S-Transferase Pi/c-Jun NH2-Terminal Kinase Complex

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Prx1 Suppresses Radiation-Induced c-Jun NH2-Terminal Kinase Signaling in Lung Cancer Cells through Interaction with the Glutathione S-Transferase Pi/c-Jun NH2-Terminal Kinase Complex

机译：PRX1通过与谷胱甘肽S-转移酶PI / C-JUM NH2-末端激酶复合酶复合酶的相互作用抑制肺癌细胞中的辐射诱导的C-Jun NH2末端激酶信号传导

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Yun-Jeong Kim; Weon-Sup Lee; Clement Ip; Ho-Zoon Chae; Eun-Mi Park; Young-Mee Park;
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1. Ligation of Major Histocompatability Complex (MHC) Class I Molecules on Human T Cells Induces Cell Death through PI-3 Kinase–induced c-Jun NH2-terminal Kinase Activity: A Novel Apoptotic Pathway Distinct from Fas-induced Apoptosis [J] . S?ren Skov, Pia Klausen, Mogens H. Claesson Journal of cell biology . 1997,第6期

机译：主要人类组织相容性复合体（MHC）I类分子在人T细胞上的连接通过PI-3激酶诱导的c-Jun NH2-末端激酶活性诱导细胞死亡：一种与Fas诱导的凋亡不同的新型凋亡途径
2. Ligation of Major Histocompatability Complex (MHC) Class I Molecules on Human T Cells Induces Cell Death through PI-3 Kinase–induced c-Jun NH2-terminal Kinase Activity: A Novel Apoptotic Pathway Distinct from Fas-induced Apoptosis [J] . S?ren Skov, Pia Klausen, Mogens H. Claesson Journal of cell biology . 1997,第6期

机译：主要人类组织相容性复合体（MHC）I类分子在人T细胞上的连接通过PI-3激酶诱导的c-Jun NH2-末端激酶活性诱导细胞死亡：一种与Fas诱导的凋亡不同的新型凋亡途径
3. Inhibition of protein kinase C α/βII and activation of c-Jun NH2-terminal kinase mediate glycyrrhetinic acid induced apoptosis in non-small cell lung cancer NCI-H460 cells [J] . SongJ., KoH.-S., SohnE.J., Bioorganic and Medicinal Chemistry Letters . 2014,第4期

机译：蛋白激酶Cα/βII的抑制和c-Jun NH2-末端激酶的激活介导甘草次酸诱导的非小细胞肺癌NCI-H460细胞凋亡
4. c-Jun N-terminal Kinase Is Involved in Transduction of Mitogenic Growth Factor Signals in Human Pancreatic Cancer Cells [C] . A.K. Winkler, Z. Liu, D. Henne-Bruns, European Society for Surgical Research . 2006

机译：C-JUM N-末端激酶参与在人胰腺癌细胞中转导的散发性生长因子信号
5. Investigation of the interaction of human Glutathione S-Transferase Pi with c-Jun N-terminal Kinase [D] . Thevenin, Anastasia Fuzaylova 2010

机译：人谷胱甘肽S-转移酶Pi与c-Jun N末端激酶相互作用的研究
6. Ligation of Major Histocompatability Complex (MHC) Class I Molecules on Human T Cells Induces Cell Death through PI-3 Kinase–induced c-Jun NH2-terminal Kinase Activity: A Novel Apoptotic Pathway Distinct from Fas-induced Apoptosis [O] . Søren Skov, Pia Klausen, Mogens H. Claesson 1997

机译：连接的主要组织相容性复合体（MHC）I类分子在人类T细胞上通过PI-3激酶诱导的c-Jun NH2-末端激酶活性诱导细胞死亡：一种与Fas诱导的凋亡不同的新型凋亡途径
7. Pharmacologic inhibitors of extracellular signal-regulated kinase (MEK/ERKs) and c-Jun NH2-terminal kinase (JNK) decrease glutathione content and sinsitize human promonocytic leukemia cells to arsenic trioxide-induced apoptosis [O] . Ramos, Adrián M., Fernández, Carlos, Esteban, Diego, 2006

机译：细胞外信号调节激酶（MEK / ERKs）和c-Jun NH2末端激酶（JNK）的药理抑制剂可降低谷胱甘肽含量并将人单核细胞白血病细胞原化为三氧化二砷诱导的凋亡

Prx1 Suppresses Radiation-Induced c-Jun NH2-Terminal Kinase Signaling in Lung Cancer Cells through Interaction with the Glutathione S-Transferase Pi/c-Jun NH2-Terminal Kinase Complex

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