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The effects of prenatal H1N1 infection at E16 on FMRP, glutamate, GABA, and reelin signaling systems in developing murine cerebellum

机译：E16对鼠霉素，谷氨酸，GABA和Reelin信号传导系统E16对E16进行产前H1N1感染的影响

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S. Hossein Fatemi; Timothy D. Folsom; Stephanie B. Liesch; Rachel E. Kneeland; Mahtab Karkhane Yousefi; Paul D. Thuras;
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1. The effects of prenatal H1N1 infection at E16 on FMRP, glutamate, GABA, and reelin signaling systems in developing murine cerebellum [J] . Fatemi S. Hossein, Folsom Timothy D., Liesch Stephanie B., Journal of Neuroscience Research . 2017,第5期

机译：E16对鼠心腺癌FMRP，谷氨酸，GABA和Reelin信号传导系统E16对E16进行蛋白质H1N1感染的影响
2. GABA receptor subunit distribution and FMRP-mGluR5 signaling abnormalities in the cerebellum of subjects with schizophrenia, mood disorders, and autism [J] . Fatemi S. Hossein, Folsom Timothy D. Schizophrenia research . 2015,第1a3期

机译：精神分裂症，情绪障碍和自闭症患者小脑中的GABA受体亚基分布和FMRP-mGluR5信号异常
3. Signal molecules and receptors in the differential development of cerebellum lobules. Acute effects of cisplatin on nitric oxide and glutamate systems in Purkinje cell population. [J] . Pisu MB, Guioli S, Conforti E, Brain research. Developmental brain research . 2003,第2期

机译：小脑小叶分化发育中的信号分子和受体。顺铂对浦肯野细胞群中一氧化氮和谷氨酸系统的急性作用。
4. A study on the effects of chronic alcohol and withdrawal on the glutamate, GABA, and dopamine systems in the basolateral amygdala of Sprague Dawley rats. [D] . Diaz, Marvin R. 2009

机译：慢性酒精和戒断对Sprague Dawley大鼠基底外侧杏仁核中谷氨酸，GABA和多巴胺系统的影响的研究。
5. The Effects of Prenatal H1N1 Infection at E16 on FMRP Glutamate GABA and Reelin Signaling Systems in Developing Murine Cerebellum [O] . S. Hossein Fatemi, Timothy D. Folsom, Stephanie B. Liesch, -1

机译：E16产前H1N1感染对发育中的小鼠小脑的FMRP谷氨酸GABA和Reelin信号传导系统的影响
6. GABA receptor subunit distribution and FMRP–mGluR5 signaling abnormalities in the cerebellum of subjects with schizophrenia, mood disorders, and autism [O] . S. Hossein Fatemi, Timothy D. Folsom 2015

机译：GABA受体亚基分布和FMRP-MGLUR5与精神分裂症，情绪障碍和自闭症的受试者小脑中的信号传导异常

The effects of prenatal H1N1 infection at E16 on FMRP, glutamate, GABA, and reelin signaling systems in developing murine cerebellum

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