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Impairments in Fast Axonal Transport and Motor Neuron Deficits in Transgenic Mice Expressing Familial Alzheimer's Disease-Linked Mutant Presenilin 1

机译：快速轴突运输和运动神经元缺陷的转基因小鼠的损伤表达家族性阿尔茨海默氏病突变体Presenilin 1

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O. Lazarov; G. A. Morfini; G. Pigino; A. Gadadhar; X. Chen; J. Robinson; H. Ho; S. T. Brady; S. S. Sisodia;
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1. Phospholipase D1 corrects impaired βAPP trafficking and neurite outgrowth in familial Alzheimer's disease-linked presenilin-1 mutant neurons [J] . Dongming Cai, Minghao Zhong, Runsheng Wang, Proceedings of the National Academy of Sciences of the United States of America . 2006,第6期

机译：磷脂酶D1纠正家族性阿尔茨海默氏病相关早老素1突变型神经元中βAPP转运受损和神经突增生
2. Impairment of axonal transport in the axon hillock and the initial segment of anterior horn neurons in transgenic mice with a G93A mutant SOD1 gene [J] . Shoichi Sasaki, Hitoshi Warita, Koji Abe, Acta Neuropathologica . 2005,第1期

机译：带有G93A突变型SOD1基因的转基因小鼠中轴突丘陵的轴突运输受损和前角神经元的初始片段
3. Bacterial artificial chromosome transgenic mice expressing a truncated mutant parkin exhibit age-dependent hypokinetic motor deficits, dopaminergic neuron degeneration, and accumulation of proteinase K-resistant alpha-synuclein. [J] . Lu XH, Fleming SM, Meurers B, The Journal of Neuroscience: The Official Journal of the Society for Neuroscience . 2009,第7期

机译：表达截短的突变体Parkin的细菌人工染色体转基因小鼠表现出年龄依赖性的运动不足，多巴胺能神经元变性和蛋白酶K抗性α-突触核蛋白的积累。
4. High content image sequence analysis for quantifying calcium signals inside cells with mutant presenilin-1 of familial alzheimer disease [C] . Fuhai Li, Xiaobo Zhou, Jinmin Zhu, IEEE/NIH Life Science Systems and Applications Workshop . 2007

机译：用突变肢体血管疾病突变血浆-1量化细胞内钙信号的高含量图像序列分析
5. Modeling clinically heterogeneous Alzheimer's disease-linked Presenilin mutations with transgenic Drosophila. [D] . Seidner, Glen Alan. 2006

机译：用转基因果蝇对临床上异质的阿尔茨海默氏病相关的早老素突变进行建模。
6. Impairments in Fast Axonal Transport and Motor Neuron Deficits in Transgenic Mice Expressing Familial Alzheimers Disease-Linked Mutant Presenilin 1 [O] . Orly Lazarov, Gerardo A. Morfini, Gustavo Pigino, 2007

机译：表达家族性阿尔茨海默氏病相关突变早老素1的转基因小鼠中快速轴突运输和运动神经元缺陷的损害。
7. Phospholipase D1 corrects impaired βAPP trafficking and neurite outgrowth in familial Alzheimer’s disease-linked presenilin-1 mutant neurons [O] . Cai, Dongming, Zhong, Minghao, Wang, Runsheng, 2006

机译：磷脂酶D1纠正家族性阿尔茨海默氏病相关早老素1突变型神经元的βAPP转运受损和神经突增生

Impairments in Fast Axonal Transport and Motor Neuron Deficits in Transgenic Mice Expressing Familial Alzheimer's Disease-Linked Mutant Presenilin 1

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