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The Cooperative Induction of Hypoxia-Inducible Factor-1α and STAT3 during Hypoxia Induced an Impairment of Tumor Susceptibility to CTL-Mediated Cell Lysis

机译：缺氧期间缺氧诱导因子-1α和Stat3的合作诱导诱导肿瘤静脉溶解损伤的肿瘤介导细胞裂解

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Muhammad Zaeem Noman; Stéphanie Buart; Jos Van Pelt; Catherine Richon; Meriem Hasmim; Nathalie Leleu; Wictoria Maria Suchorska; Abdelali Jalil; Yann Lecluse; Faten El Hage; Massimo Giuliani; Christophe Pichon; Bruno Azzarone; Nathalie Mazure; Pedro Romero; Fathia Mami-Chouaib; Salem Chouaib;
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1. The cooperative induction of hypoxia-inducible factor-1alpha and STAT3 during hypoxia induced an impairment of tumor susceptibility to CTL-mediated cell lysis. [J] . Noman MZ, Buart S, Van-Pelt J, The Journal of Immunology: Official Journal of the American Association of Immunologists . 2009,第6期

机译：缺氧过程中缺氧诱导因子-1α和STAT3的协同诱导导致肿瘤对CTL介导的细胞溶解的敏感性降低。
2. The cooperative induction of hypoxia-inducible factor-1alpha and STAT3 during hypoxia induced an impairment of tumor susceptibility to CTL-mediated cell lysis. [J] . Noman MZ, Buart S, Van-Pelt J, The Journal of Immunology: Official Journal of the American Association of Immunologists . 2009,第6期

机译：缺氧过程中缺氧诱导因子-1α和STAT3的协同诱导导致肿瘤对CTL介导的细胞溶解的敏感性降低。
3. Hypoxia-dependent inhibition of tumor cell susceptibility to CTL-mediated lysis involves NANOG induction in target cells. [J] . Hasmim M, Noman MZ, Lauriol J, The Journal of Immunology: Official Journal of the American Association of Immunologists . 2011,第8期

机译：缺氧依赖性抑制肿瘤细胞对CTL介导的裂解的敏感性涉及在靶细胞中诱导NANOG。
4. Therapeutic Silencing of Hypoxia-Inducible Factor-1α Using Mixed Micelle Particles Inhibits Prostate Cancer Cell Growth under Hypoxia [C] . Xi-Qiu Liu, Meng-Hua Xiong, Jun Wang World biomaterials congress . 2012

机译：混合胶束粒子对缺氧诱导因子-1α的治疗沉默抑制缺氧条件下前列腺癌细胞的生长
5. Physical Activity Optimizes Circuit-Specific Cellular Metabolism in Neuroplasticity: A Role for Hypoxia-Inducible Factor-1 and its Downstream Targets [D] . Halliday, Matthew Ryan. 2019

机译：物理活性优化神经塑性中的电路特异性细胞代谢：缺氧诱导因子-1的作用及其下游靶标
6. Celastrol Stimulates Hypoxia-Inducible Factor-1 Activity in Tumor Cells by Initiating the ROS/Akt/p70S6K Signaling Pathway and Enhancing Hypoxia-Inducible Factor-1α Protein Synthesis [O] . Xiaoxi Han, Shengkun Sun, Ming Zhao, -1

机译：Celastrol通过启动ROS / Akt / p70S6K信号通路并增强缺氧诱导因子1α蛋白合成来刺激肿瘤细胞中的缺氧诱导因子1活性。
7. Hypoxia-dependent inhibition of tumor cell susceptibility to CTL-mediated lysis involves NANOG induction in target cells. [O] . Hasmim, Meriem, Noman, Muhammad Zaeem, Lauriol, Jessica, 2011

机译：缺氧依赖性抑制肿瘤细胞对CTL介导的裂解的敏感性涉及在靶细胞中诱导NANOG。

The Cooperative Induction of Hypoxia-Inducible Factor-1α and STAT3 during Hypoxia Induced an Impairment of Tumor Susceptibility to CTL-Mediated Cell Lysis

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