首页> 外文OA文献 >Defect in the Induction of Nitric Oxide Synthase by Lipopolysaccharide (LPS) in an LPS-Resistant Mutant of a Murine Macrophage-Like Cell Line, J774.1.

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Defect in the Induction of Nitric Oxide Synthase by Lipopolysaccharide (LPS) in an LPS-Resistant Mutant of a Murine Macrophage-Like Cell Line, J774.1.

机译：脂多糖（LPS）在鼠巨噬细胞样细胞系的LPS抗突变体中诱导一氧化氮合酶的缺陷，J774.1。

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Tetsuji NODA; Fumio AMANO;
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年度 1998
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1. Defect in the induction of nitric oxide synthase by lipopolysaccharide (LPS) in an LPS-resistant mutant of a murine macrophage-like cell line, J774.1. [J] . Noda T, Amano F Biological & pharmaceutical bulletin . 1998,第7期

机译：小鼠巨噬细胞样细胞系J774.1的LPS抗性突变体中的脂多糖（LPS）诱导一氧化氮合酶诱导的缺陷。
2. Structure-activity relationships of lipopolysaccharide (LPS) in tumor necrosis factor-alpha (TNF-alpha) production and induction of macrophage cell death in the presence of cycloheximide (CHX) in a murine macrophage-like cell line, J774.1. [J] . Karahashi H, Amano F Biological & pharmaceutical bulletin . 1998,第10期

机译：在鼠类巨噬细胞样细胞系J774.1中，在存在环己酰亚胺（CHX）的情况下，肿瘤坏死因子-α（TNF-α）产生和巨噬细胞死亡诱导中的脂多糖（LPS）的结构活性关系。
3. Difference in Legionella pneumophila growth permissiveness between J774.1 murine macrophage-like JA-4 cells and lipopolysaccharide (LPS)-resistant mutant cells, LPS1916, after stimulation with LPS. [J] . F Kura, K Suzuki, H Watanabe, Infection and immunity . 1994,第12期

机译：用LPS刺激后，J774.1鼠巨噬细胞样JA-4细胞与抗脂多糖（LPS）的突变细胞LPS1916之间的嗜肺军团菌生长许可性有所不同。
4. Induction of TNF-alpha, IL-6 and Nitric Oxide by Titanium Nitride, Titanium Silicide and Titanium Telluride Particles and LPS in RAW 264.7 Murine Macrophage Cells [C] . V.M. Hitchins, A.R. Mtungwa, K. Merritt Annual meeting of the Society For Biomaterials . 2003

机译：氮化钛，硅化钛和碲化钛颗粒以及LPS在RAW 264.7小鼠巨噬细胞中诱导TNF-α，IL-6和一氧化氮
5. Effects of nitric oxide selectively derived from macrophage inducible nitric oxide synthase in murine septic lung injury [D] . Farley, Kalamo Shane 2008

机译：巨噬细胞诱导型一氧化氮合酶选择性衍生的一氧化氮在小鼠败血症性肺损伤中的作用
6. Relationship of tumor necrosis factor alpha the nitric oxide synthase pathway and lipopolysaccharide to the killing of gamma interferon-treated macrophage-like RAW264.7 cells by Rickettsia prowazekii. [O] . J Turco, H H Winkler 1994

机译：肿瘤坏死因子α一氧化氮合酶途径和脂多糖与丙种立克次体杀死γ-干扰素处理的巨噬细胞样RAW264.7细胞的关系。
7. Molecular cloning of cDNA for nonhepatic mitochondrial arginase (arginase II) and comparison of its induction with nitric oxide synthase in a murine macrophage-like cell line [O] . Gotoh Tomomi, Sonoki Takashi, Nagasaki Akitoshi, 1996

机译：小鼠巨噬细胞样细胞系中非肝线粒体精氨酸酶（精氨酸酶II）cDNA的分子克隆及其与一氧化氮合酶诱导的比较
8. PKC Mediates LPS- and Phorbol-Induced Cardiac Cell Nitric Oxide Synthase Activityand Hypocontractility [R] . McKenna, T. M., Li, S., Tao, S. 1995

机译：pKC介导Lps和phorbol诱导的心肌细胞一氧化氮合酶活性和低缩量

Defect in the Induction of Nitric Oxide Synthase by Lipopolysaccharide (LPS) in an LPS-Resistant Mutant of a Murine Macrophage-Like Cell Line, J774.1.

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