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A new mouse line with reduced GluA2 Q/R site RNA editing exhibits loss of dendritic spines, hippocampal CA1-neuron loss, learning and memory impairments and NMDA receptor-independent seizure vulnerability

机译：一种新的鼠标线，具有缩减Glua2 Q / R立网RNA编辑，表现出树突状脊柱的丧失，海马CA1-Neuron丢失，学习和记忆障碍和NMDA受体独立的癫痫发作

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Lyndsey M. Konen; Amanda L. Wright; Gordon A. Royle; Gary P. Morris; Benjamin K. Lau; Patrick W. Seow; Raphael Zinn; Luke T. Milham; Christopher W. Vaughan; Bryce Vissel;
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年度 2020
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入库时间 2022-08-20 22:14:23

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1. A new mouse line with reduced GluA2 Q/R site RNA editing exhibits loss of dendritic spines, hippocampal CA1-neuron loss, learning and memory impairments and NMDA receptor-independent seizure vulnerability [J] . Lyndsey M. Konen, Amanda L. Wright, Gordon A. Royle, Molecular brain . 2020,第1期

机译：一种新的鼠标线，具有缩减Glua2 Q / R立网RNA编辑，表现出树突状脊柱的丧失，海马CA1-Neuron丢失，学习和记忆障碍和NMDA受体独立的癫痫发作
2. Loss of GluN2B-containing NMDA receptors in CA1 hippocampus and cortex impairs long-term depression, reduces dendritic spine density, and disrupts learning. [J] . Brigman JL, Wright T, Talani G, The Journal of Neuroscience: The Official Journal of the Society for Neuroscience . 2010,第13期

机译：CA1海马和皮层中含有GluN2B的NMDA受体的丢失损害了长期的抑郁症，降低了树突状脊柱的密度，并破坏了学习。
3. Vitamin B1-deficient mice show impairment of hippocampus-dependent memory formation and loss of hippocampal neurons and dendritic spines: potential microendophenotypes of Wernicke-Korsakoff syndrome [J] . Inaba Hiroyoshi, Kishimoto Takuya, Oishi Satoru, Bioscience, Biotechnology, and Biochemistry . 2016,第12期

机译：维生素B1缺陷小鼠表现出海马依赖性记忆形成和海马神经元和树突脊柱损失的损害：Wernicke-Korsakoff综合征的潜在微生物术
4. A new mouse line with reduced GluA2 Q/R site RNA editing exhibits loss of dendritic spines hippocampal CA1-neuron loss learning and memory impairments and NMDA receptor-independent seizure vulnerability [O] . Lyndsey M. Konen, Amanda L. Wright, Gordon A. Royle, 2020

机译：具有减少的GluA2 Q / R位点RNA编辑的新小鼠系表现出树突棘丧失海马CA1-神经元丧失学习和记忆障碍以及NMDA受体非依赖性癫痫发作易感性
5. Loss of GluN2B-containing NMDA receptors in CA1 hippocampus and cortex impairs long-term depression, reduces dendritic spine density, and disrupts learning [O] . BRIGMAN JL, WRIGHT T, TALANI G, 2010

机译：CA1海马和皮层中含有GluN2B的NMDA受体的丢失损害了长期的抑郁症，降低了树突状脊柱的密度，并破坏了学习

A new mouse line with reduced GluA2 Q/R site RNA editing exhibits loss of dendritic spines, hippocampal CA1-neuron loss, learning and memory impairments and NMDA receptor-independent seizure vulnerability

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