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Neuronal Deficiency of Presenilin 1 Inhibits Amyloid Plaque Formation and Corrects Hippocampal Long-Term Potentiation But Not a Cognitive Defect of Amyloid Precursor Protein V717I Transgenic Mice

机译：Presenilin 1的神经元缺乏抑制淀粉样蛋白斑块形成并校正海马长期增强，但不是淀粉样蛋白前体蛋白V717I转基因小鼠的认知缺陷

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Ilse Dewachter; Delphine Reversé; Nathalie Caluwaerts; Laurence Ris; Cuno Kuipéri; Chris Van den Haute; Kurt Spittaels; Lieve Umans; Lutgarde Serneels; Els Thiry; Dieder Moechars; Mark Mercken; Emile Godaux; Fred Van Leuven;
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年度 2002
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1. Neuronal deficiency of presenilin 1 inhibits amyloid plaque formation and corrects hippocampal long-term potentiation but not a cognitive defect of amyloid precursor protein (V717I) transgenic mice. [J] . Dewachter I, Reverse D, Caluwaerts N, The Journal of Neuroscience: The Official Journal of the Society for Neuroscience . 2002,第9期

机译：早老素1的神经元缺乏抑制淀粉样斑块的形成并纠正海马长期增强，但不是淀粉样前体蛋白（V717I）转基因小鼠的认知缺陷。
2. A study of long-term potentiation in transgenic mice over-expressing mutant forms of both amyloid precursor protein and presenilin-1 [J] . Stephen M Fitzjohn, Frederick Kuenzi, Robin A Morton, Molecular brain . 2010,第1期

机译：过度表达淀粉样蛋白前体蛋白和早老素-1突变形式的转基因小鼠中的长期增强作用的研究
3. Neprilysin overexpression inhibits plaque formation but fails to reduce pathogenic Abeta oligomers and associated cognitive deficits in human amyloid precursor protein transgenic mice. [J] . Meilandt WJ, Cisse M, Ho K, The Journal of Neuroscience: The Official Journal of the Society for Neuroscience . 2009,第7期

机译：中性溶酶的过表达抑制了斑块的形成，但未能减少人类淀粉样蛋白前体蛋白转基因小鼠中的致病性Abeta低聚物和相关的认知缺陷。
4. Proteomic Imaging of amyloids in Brains from Amyloid Precursor Protein (APP) Transgenic Mice in comparison with Human Alzheimer Amyloid [C] . Masaya Ikegawa, Tomohiro Miyasaka, Noriyuki Iwasaki, ASMS Conference on Mass Spectrometry and Allied Topics . 2015

机译：淀粉样蛋白前体蛋白（APP）转基团的淀粉样蛋白淀粉样蛋白蛋白质组学成像与人阿尔茨海默蛋白相比
5. Dictyostelium discoideum possess a highly diverged presenilin/gamma-secretase that accurately processes human amyloid precursor protein and is required for growth and correct cell fate determination. [D] . McMains, Vanessa C. 2010

机译：盘基网柄菌拥有高度分化的早老素/γ-分泌酶，可准确加工人淀粉样蛋白前体蛋白，是生长和正确确定细胞命运所必需的。
6. Neuronal Deficiency of Presenilin 1 Inhibits Amyloid Plaque Formation and Corrects Hippocampal Long-Term Potentiation But Not a Cognitive Defect of Amyloid Precursor Protein V717I Transgenic Mice [O] . Ilse Dewachter, Delphine Reversé, Nathalie Caluwaerts, 2002

机译：早老素1的神经元缺乏抑制淀粉样斑块形成并纠正海马长期增强但不是淀粉样前体蛋白V717I转基因小鼠的认知缺陷。
7. A study of long-term potentiation in transgenic mice over-expressing mutant forms of both amyloid precursor protein and presenilin-1 [O] . Fitzjohn Stephen M., Kuenzi Frederick, Morton Robin A., 2011

机译：过度表达淀粉样蛋白前体蛋白和早老素-1突变形式的转基因小鼠中的长期增强作用的研究

Neuronal Deficiency of Presenilin 1 Inhibits Amyloid Plaque Formation and Corrects Hippocampal Long-Term Potentiation But Not a Cognitive Defect of Amyloid Precursor Protein V717I Transgenic Mice

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