首页> 外文OA文献 >DNA Polymerase Iota (Pol ι) Promotes Radioresistance of Esophageal Squamous Cell Carcinoma through Blocking Ubiquitin-mediated RAD51 Degradation and Homologous Recombinational Repair after Radiation-induced DNA Damage

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DNA Polymerase Iota (Pol ι) Promotes Radioresistance of Esophageal Squamous Cell Carcinoma through Blocking Ubiquitin-mediated RAD51 Degradation and Homologous Recombinational Repair after Radiation-induced DNA Damage

机译：DNA聚合酶IOTA（POL 1）通过阻断遍突蛋白介导的RAD51降解和辐射诱导的DNA损伤后的同源重组修复来促进食管鳞状细胞癌的辐射敏感度

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J. Zhou; X. Li; Z. Shang; A. Gao; S. Ji; C. He;
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1. Overexpression of DNA polymerase iota (Polι) in esophageal squamous cell carcinoma [J] . Fang Xie, Jianping Cao, Jinchang Wu, Cancer science. . 2012,第8期

机译：食管鳞状细胞癌中DNA聚合酶Iota（Polι）的过表达
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3. The Cancer/Testes (CT) Antigen HORMAD1 promotes Homologous Recombinational DNA Repair and Radioresistance in Lung adenocarcinoma cells [J] . Yanzhe Gao, Jordan Kardos, Yang Yang, Scientific reports. . 2018,第1期

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4. Upregulation of DNA polymerase iota (Pol ι) in esophageal squamous cell carcinoma [C] . Jundong Zhou, Shuyu Zhang, Xie lin, 2011 Asia-Pacific Conference of tumor biology and medicine . 2011

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5. The versatile role of homologous recombination in plant cell: Repair of DNA damage, stress-directed genome evolution and foreign DNA integration. [D] . Boyko, Oleksandr. 2008

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7. Cancer-associated Fibroblast–promoted LncRNA DNM3OS Confers Radioresistance by Regulating DNA Damage Response in Esophageal Squamous Cell Carcinoma [O] . Hongfang Zhang, Yuhui Hua, Zhenzhen Jiang, 2018

机译：癌症相关的成纤维细胞促进的LNCRNA DNM3OS通过调节食管鳞状细胞癌中的DNA损伤反应来赋予辐射血管

DNA Polymerase Iota (Pol ι) Promotes Radioresistance of Esophageal Squamous Cell Carcinoma through Blocking Ubiquitin-mediated RAD51 Degradation and Homologous Recombinational Repair after Radiation-induced DNA Damage

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