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Indexes of citrulline metabolism in rat liver under the toxic injury against the background of alimentary protein deficiency

机译：在消化蛋白质缺陷背景下毒性损伤瘤肝粉煤中酸瘤代谢指标

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H. P. Kopylchuk; I. M. Nykolaichuk; I. S. Lylyk;
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年度 2020
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1. Indexes of citrulline metabolism in rat liver under the toxic injury against the background of alimentary protein deficiency [J] . H. P. Kopylchuk, I. M. Nykolaichuk, I. S. Lylyk Ukrainian Biochemical Journal . 2020,第1期

机译：在消化蛋白质缺陷背景下毒性损伤瘤肝粉煤中酸瘤代谢指标
2. NADH:ubiquinone reductase and succinate dehydrogenase activity in the liver of rats with acetaminophen-induced toxic hepatitis on the background of alimentary protein deficiency [J] . G. P. Kopylchuk, O. M. Voloshchuk Ukrainian Biochemical Journal . 2015,第1期

机译：缺乏蛋白质的背景下对乙酰氨基酚诱发的中毒性肝炎大鼠肝脏NADH：泛醌还原酶和琥珀酸脱氢酶活性
3. Peculiarities of the free radical processes in rat liver mitochondria under toxic hepatitis on the background of alimentary protein deficiency [J] . G. P. Kopylchuk, O. M. Voloshchuk Ukrainian Biochemical Journal . 2016,第2期

机译：蛋白质缺乏的背景下中毒性肝炎大鼠肝线粒体自由基过程的特殊性
4. Evaluation of the bone tissue mechanical parameters after induced alimentary Cu-deficiency followed by supplementary injection of Cu nanoparticles in rats [C] . O Sachenkov, L Kharislamova, N Shamsutdinova International Youth Confernece on Interdisciplinary Problems of Nanotechnology, Biomedicine and Nanotoxicology . 2016

机译：诱导消毒Cu缺乏后骨组织机械参数的评价，然后用大鼠Cu纳米粒子的补充注射
5. Mechanisms of sulindac/LPS-induced liver injury in rats: An animal model of drug-induced idiosyncratic hepatotoxicity. [D] . Zou, Wei. 2010

机译：舒林酸/ LPS诱导的大鼠肝损伤的机制：药物诱导的特异性肝毒性的动物模型。
6. Toxic liver injury. Inhibition by dimethylnitrosamine of incorporation of labelled amino acids into proteins of rat-liver preparations in vitro [O] . T. Hultin, E. Arrhenius, H. Löw, 1960

机译：中毒性肝损伤。二甲基亚硝胺抑制标记的氨基酸体外掺入大鼠肝制剂的蛋白质
7. Copper metabolism in rats under toxic injury by acetaminophen on the background of alimentary protein deficiency [O] . O. M. Voloshchuk, G. P. Kopylchuk 2018

机译：乙酰苯乙酰胺毒性毒性血液损伤的铜代谢

Indexes of citrulline metabolism in rat liver under the toxic injury against the background of alimentary protein deficiency

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