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Quantitative Relationships between Huntingtin Levels, Polyglutamine Length, Inclusion Body Formation, and Neuronal Death Provide Novel Insight into Huntington's Disease Molecular Pathogenesis

机译：亨廷顿水平，聚谷氨酰胺长度，包容体形成和神经元死亡之间的定量关系为亨廷顿疾病分子发病机构提供了新的洞察力

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J. Miller; M. Arrasate; B. A. Shaby; S. Mitra; E. Masliah; S. Finkbeiner;
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1. Quantitative relationships between huntingtin levels, polyglutamine length, inclusion body formation, and neuronal death provide novel insight into huntington's disease molecular pathogenesis. [J] . Miller J, Arrasate M, Shaby BA, The Journal of Neuroscience: The Official Journal of the Society for Neuroscience . 2010,第31期

机译：亨廷顿蛋白水平，聚谷氨酰胺长度，包涵体形成和神经元死亡之间的定量关系为亨廷顿病分子发病机理提供了新颖的见解。
2. Quantitative relationships between huntingtin levels, polyglutamine length, inclusion body formation, and neuronal death provide novel insight into huntington's disease molecular pathogenesis. [J] . Miller J, Arrasate M, Shaby BA, The Journal of Neuroscience: The Official Journal of the Society for Neuroscience . 2010,第31期

机译：亨廷顿蛋白水平，聚谷氨酰胺长度，包涵体形成和神经元死亡之间的定量关系为亨廷顿病分子发病机理提供了新颖的见解。
3. Huntingtin Spheroids and Protofibrils as Precursors in Polyglutamine Fibrilization;Inclusion Body Formation Reduces Levels of Mutant Huntingtin and the Risk of Neuronal Death;Pivotal Role of Oligomerization in Expanded Polyglutamine Neurodegenerative [J] . Albert R.La Spada Chemtracts . 2005,第3期

机译：亨廷顿蛋白球体和原纤维作为聚谷氨酰胺原纤维化的前体;包涵体形成降低了亨廷顿蛋白突变体的水平和神经元死亡的风险;寡聚化在扩大的聚谷氨酰胺神经变性中的关键作用
4. Quantitative Relationships between Huntingtin Levels Polyglutamine Length Inclusion Body Formation and Neuronal Death Provide Novel Insight into Huntingtons Disease Molecular Pathogenesis [O] . Jason Miller, Montserrat Arrasate, Benjamin A. Shaby, 2010

机译：亨廷顿水平聚谷氨酰胺长度包涵体形成和神经元死亡之间的定量关系提供了新的洞察亨廷顿氏病分子发病机理
5. Inclusion body formation reduces levels of mutant huntingtin and the risk of neuronal death [O] . Montserrat Arrasate, Siddhartha Mitra, Erik S. Schweitzer, 2004

机译：包含体形成减少了突变亨廷顿的水平和神经元死亡的风险

Quantitative Relationships between Huntingtin Levels, Polyglutamine Length, Inclusion Body Formation, and Neuronal Death Provide Novel Insight into Huntington's Disease Molecular Pathogenesis

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