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Genetic deletion of the MT1 or MT2 melatonin receptors abrogates methamphetamine-induced reward in C3H/HeN mice

机译：MT1或MT2褪黑素受体的遗传缺失废除C3H /母鸡小鼠中甲基苯丙胺诱导的奖励

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Shannon J. Clough; Anthony J. Hutchinson; Randall L. Hudson; Margarita L. Dubocovich;
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1. Food-induced reinforcement is abrogated by the genetic deletion of the MT1 or MT2 melatonin receptor in C3H/HeN mice [J] . Clough Shannon J., Hudson Randall L., Dubocovich Margarita L. Behavioural Brain Research: An International Journal . 2018,第期

机译：通过C3H /母鸡小鼠的MT1或MT2褪黑激素受体的遗传缺失废除食物诱导的抗粘度
2. Food-induced reinforcement is abrogated by the genetic deletion of the MT1 or MT2 melatonin receptor in C3H/HeN mice [J] . Clough Shannon J., Hudson Randall L., Dubocovich Margarita L. Behavioural Brain Research: An International Journal . 2018,第期

机译：通过C3H /母鸡小鼠的MT1或MT2褪黑激素受体的遗传缺失废除食物诱导的抗粘度
3. Genetic deletion of the MT_1 or MT_2 melatonin receptors abrogates methamphetamine-induced reward in C3H/HeN mice [J] . Shannon J. Clough, Anthony J. Hutchinson, Rall L Hudson, Physiology & behavior . 2014,第Null期

机译：MT_1或MT_2褪黑激素受体的基因缺失消除了甲基苯丙胺对C3H / HeN小鼠的奖励
4. The Role of Melatonin Receptor IB Gene (MTNR1B) in Adolescent Idiopathic Scoliosis - A Genetic Association Study [C] . QIU XS, TANG NL, YEUNG HY, International Research Society of Spinal Deformities . 2008

机译：褪黑激素受体IB基因（MTNR1B）在青少年特发性脊柱侧凸的作用 - 一种遗传结合研究
5. Melatonin Potentiates Running Induced Neurogenesis in the Dentate Gyrus of Adult C3H/HeN mice. [D] . Liu, Jiabei JJ. 2011

机译：褪黑素增强了成年C3H / HeN小鼠齿状回中奔跑诱导的神经发生。
6. Genetic Deletion of the MT1 or MT2 Melatonin Receptors Abrogates Methamphetamine-Induced Reward in C3H/HeN Mice [O] . Shannon J. Clough, Anthony J. Hutchinson, Randall L. Hudson, -1

机译：MT1或MT2褪黑素受体的基因删除废除了甲基苯丙胺诱导的C3H / HeN小鼠奖励
7. Food-induced reinforcement is abrogated by the genetic deletion of the MT 1 or MT 2 melatonin receptor in C3H/HeN mice [O] . Shannon J. Clough, Randall L. Hudson, Margarita L. Dubocovich 2018

机译：通过C3H /母鸡小鼠的MT 1或MT 2褪黑激素受体的遗传缺失废除食物诱导的增强物

Genetic deletion of the MT1 or MT2 melatonin receptors abrogates methamphetamine-induced reward in C3H/HeN mice

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