首页> 外文OA文献 >MK-2206 co-treatment with 5-fluorouracil or doxorubicin enhances chemosensitivity and apoptosis in gastric cancer by attenuation of Akt phosphorylation

【2h】

MK-2206 co-treatment with 5-fluorouracil or doxorubicin enhances chemosensitivity and apoptosis in gastric cancer by attenuation of Akt phosphorylation

机译：用5-氟尿嘧啶或多柔比星进行合作，通过衰减Akt磷酸化增强胃癌中的化学敏感性和凋亡

代理获取

本网站仅为用户提供外文OA文献查询和代理获取服务，本网站没有原文。下单后我们将采用程序或人工为您竭诚获取高质量的原文，但由于OA文献来源多样且变更频繁，仍可能出现获取不到、文献不完整或与标题不符等情况，如果获取不到我们将提供退款服务。请知悉。

页面导航

著录项
引文网络
相似文献
相关主题

著录项

作者
Piaopiao Jin; Chi-Chun Wong; Sibin Mei; Xiangkang He; Yun Qian; Leimin Sun;
展开▼
作者单位

展开▼
年度 2016
总页数
原文格式 PDF
正文语种
中图分类

相似文献

外文文献
中文文献
专利

1. MK-2206 co-treatment with 5-fluorouracil or doxorubicin enhances chemosensitivity and apoptosis in gastric cancer by attenuation of Akt phosphorylation [J] . Piaopiao Jin, Chi Chun Wong, Sibin Mei, OncoTargets and therapy . 2016,第4期

机译：MK-2206与5-氟尿嘧啶或阿霉素共同治疗可通过减弱Akt磷酸化作用增强胃癌的化学敏感性和细胞凋亡
2. CISD2 enhances the chemosensitivity of gastric cancer through the enhancement of 5-FU-induced apoptosis and the inhibition of autophagy by AKT/mTOR pathway [J] . Elisabete de Fátima Pólo de Almeida Nunes, Yingming Jiang, Peter V. Kochunov, Cancer Medicine . 2017,第10期

机译：CISD2通过增强5-FU诱导的细胞凋亡和通过AKT / mTOR途径抑制自噬来增强胃癌的化学敏感性
3. Parameritannin A-2 from Urceola huaitingii enhances doxorubicin-induced mitochondria-dependent apoptosis by inhibiting the PI3K/Akt, ERK1/2 and p38 pathways in gastric cancer cells [J] . Chemico-biological interactions . 2020,第期

机译：来自Urceola Huaitingii的Parameritannin A-2通过抑制胃癌细胞中的PI3K / AKT，ERK1 / 2和P38途径来增强多柔比星诱导的线粒体依赖性细胞凋亡
4. Theranostic pH responsive doxorubicin loaded nanoparticles inducing active targeting and apoptosis for advanced gastric cancer [C] . Malcolm Xing World biomaterials congress . 2016

机译：pH敏感的阿洛霉素纳米粒的催化活性靶向和凋亡治疗晚期胃癌。
5. Phosphorylation of NF-kappaB RelA/p65 on Ser536 signals cancer cells to death and enhances chemosensitivity. [D] . Bu, Yiwen. 2014

机译：Ser536上的NF-κBRelA / p65的磷酸化信号表明癌细胞死亡，并增强了化学敏感性。
6. MK-2206 co-treatment with 5-fluorouracil or doxorubicin enhances chemosensitivity and apoptosis in gastric cancer by attenuation of Akt phosphorylation [O] . Piaopiao Jin, Chi Chun Wong, Sibin Mei, 2016

机译：MK-2206与5-氟尿嘧啶或阿霉素共同治疗可通过减弱Akt磷酸化来增强胃癌的化学敏感性和细胞凋亡
7. Co-treatment of LY294002 or MK-2206 with AZD5363 Attenuates AZD5363-induced Increase in the Level of Phosphorylated AKT [O] . AE-RAN CHOI, JU-HWA KIM, YEON HWA WOO, 2016

机译：使用AZD5363的LY294002或MK-2206的合作衰减AZD5363诱导的磷酸化AKT水平增加

MK-2206 co-treatment with 5-fluorouracil or doxorubicin enhances chemosensitivity and apoptosis in gastric cancer by attenuation of Akt phosphorylation

著录项

引文网络

相似文献

相关主题

期刊订阅