首页> 外文OA文献 >SMNΔ7, the major product of the centromeric survival motor neuron (SMN2) gene, extends survival in mice with spinal muscular atrophy and associates with full-length SMN

【2h】

SMNΔ7, the major product of the centromeric survival motor neuron (SMN2) gene, extends survival in mice with spinal muscular atrophy and associates with full-length SMN

机译：SMNΔ7，浓缩级麦克肌腱（SMN2）基因的主要产物，延长了小鼠的生存，脊柱肌肉萎缩和全长SMN缔合

代理获取

本网站仅为用户提供外文OA文献查询和代理获取服务，本网站没有原文。下单后我们将采用程序或人工为您竭诚获取高质量的原文，但由于OA文献来源多样且变更频繁，仍可能出现获取不到、文献不完整或与标题不符等情况，如果获取不到我们将提供退款服务。请知悉。

页面导航

著录项
引文网络
相似文献
相关主题

著录项

作者
Thanh T. Le; Lan T. Pham; Matthew E.R. Butchbach; Honglai L. Zhang; Umrao R. Monani; Daniel D. Coovert; Tatiana O. Gavrilina; Lei Xing; Gary J. Bassell; Arthur H.M. Burghes;
展开▼
作者单位

展开▼
年度 2005
总页数
原文格式 PDF
正文语种
中图分类

相似文献

外文文献
中文文献
专利

1. SMN{Delta}7, the major product of the centromeric survival motor neuron (SMN2) gene, extends survival in mice with spinal muscular atrophy and associates with full-length SMN. [J] . Le TT, Pham LT, Butchbach ME, Human Molecular Genetics . 2005,第6期

机译：SMN {Delta} 7是着丝粒存活运动神经元（SMN2）基因的主要产物，可延长脊髓性肌萎缩症小鼠的存活率，并与全长SMN相关。
2. Reduced survival of motor neuron (SMN) protein in motor neuronal progenitors functions cell autonomously to cause spinal muscular atrophy in model mice expressing the human centromeric (SMN2) gene. [J] . Park GH, Maeno Hikichi Y, Awano T, The Journal of Neuroscience: The Official Journal of the Society for Neuroscience . 2010,第36期

机译：运动神经元祖细胞中运动神经元（SMN）蛋白存活率的降低会自动发挥功能，导致表达人类着丝粒（SMN2）基因的模型小鼠的脊髓肌萎缩。
3. The human centromeric survival motor neuron gene (SMN2) rescues embryonic lethality in Smn?/? mice and results in a mouse with spinal muscular atrophy [J] . Umrao R. Monani Michael Sendtner Daniel D. Coovert D. William Parsons Catia Andreassi Thanh T. Le Sibylle Jablonka Berthold Schrank Wilfried Rossoll Thomas W. Prior Glenn E. Morris and Arthur H.M. Burghes Human Molecular Genetics . 2007,第21期

机译：人类着丝粒存活运动神经元基因（SMN2）挽救Smn ？/？小鼠的胚胎致死率，并导致小鼠脊髓性肌萎缩
4. Early decrease of survival signal protein and DNA repair enzyme in spinal motor neuron of presymptomatic transgenic mice with a mutant SOD1 gene [C] . M. Shiote, H. Ilieva, I. Nagano, International Symposium on Molecular Mechanism and Epochal Therapeutics for Ischemic Stroke and Dementia . 2003

机译：用突变SOD1基因早期降低生存信号蛋白和脊柱运动神经元中的脊髓运动神经元的DNA修复酶
5. Targeted gene alteration in SMA patient cells: Genetic conversion of an SMN2 gene to SMN1 increases full-length SMN production. [D] . Callahan, Stephanie. 2009

机译：SMA患者细胞中的靶向基因改变：SMN2基因向SMN1的遗传转化增加了全长SMN的产生。
6. Reduced Survival of Motor Neuron (SMN) Protein in Motor Neuronal Progenitors Functions Cell Autonomously to Cause Spinal Muscular Atrophy in Model Mice Expressing the Human Centromeric (SMN2) Gene [O] . Gyu-Hwan Park, Yuka Maeno-Hikichi, Tomoyuki Awano, 2010

机译：运动神经元（SMN）蛋白在运动神经元祖细胞中的功能自主减少从而导致表达人着丝粒（SMN2）基因的小鼠模型中的脊髓性肌萎缩症的存活率降低
7. The human centromeric survival motor neuron gene (SMN2) rescues embryonic lethality in Smn-/- mice and results in a mouse with spinal muscular atrophy [O] . U. R. Monani 2000

机译：人体浓缩运动Moton神经元基因（SMN2）在SMN / - 小鼠中拯救胚胎致死态，并导致脊柱肌萎缩的小鼠

SMNΔ7, the major product of the centromeric survival motor neuron (SMN2) gene, extends survival in mice with spinal muscular atrophy and associates with full-length SMN

著录项

引文网络

相似文献

相关主题

期刊订阅