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Author Correction: Defects in mtDNA replication challenge nuclear genome stability through nucleotide depletion and provide a unifying mechanism for mouse progerias

机译：作者校正：MTDNA复制中的缺陷通过核苷酸耗尽来挑战核基因组稳定性，并为小鼠普罗格里亚提供统一机制

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Riikka H. Hämäläinen; Juan C. Landoni; Kati J. Ahlqvist; Steffi Goffart; Sanna Ryytty; M. Obaidur Rahman; Virginia Brilhante; Katherine Icay; Sampsa Hautaniemi; Liya Wang; Marikki Laiho; Anu Suomalainen;
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1. Author Correction: MitoTALEN reduces mutant mtDNA load and restores tRNA Ala levels in a mouse model of heteroplasmic mtDNA mutation. [J] . Sandra R Bacman, Johanna H K Kauppila, Claudia V Pereira, Nature medicine . 2018,第12期

机译：作者校正：Mitotalen在异质MTDNA突变的小鼠模型中减少突变体MTDNA载荷并恢复TRNA ALA水平。
2. DNA repair defects and genome instability in Hutchinson-Gilford Progeria Syndrome [J] . Gonzalo Susana, Kreienkamp Ray Current Opinion in Cell Biology . 2015,第Null期

机译：Hutchinson-Gilford早衰综合征的DNA修复缺陷和基因组不稳定
3. Mitochondrial Genome Instability and mtDNA Depletion in Human Cancers [J] . HSIN-CHEN LEE, PEN-HUI YIN, JIN-CHING LIN, Annals of the New York Academy of Sciences . 2005,第May期

机译：线粒体基因组不稳定性和人类癌症中的mtDNA消耗
4. Mitochondrial Genome Instability and mtDNA Depletion in Human Cancers [C] . HSIN-CHEN LEE, PEN-HUI YIN, JIN-CHING LIN, Asian Society for Mitochondrial Research and Medicine . 2005

机译：人类癌症中的线粒体基因组不稳定性和MTDNA枯竭
5. Uncovering novel and unexpected mechanisms for maintaining genome stability during DNA replication [D] . Smith, Kevin D. 2011

机译：发现在DNA复制过程中维持基因组稳定性的新颖机制
6. Correction: Correction: Single Nucleotide Polymorphisms Reveal Genetic Structuring of the Carpathian Newt and Provide Evidence of Interspecific Gene Flow in the Nuclear Genome [O] . -1

机译：校正：校正：单核苷酸多态性揭示了喀尔巴阡New的遗传结构并提供了核基因组中种间基因流动的证据。
7. Correction: Correction: Single Nucleotide Polymorphisms Reveal Genetic Structuring of the Carpathian Newt and Provide Evidence of Interspecific Gene Flow in the Nuclear Genome [O] . 2014

机译：校正：校正：单核苷酸多态性揭示喀尔巴阡植物蝾螈的遗传结构，并提供核基因组中三种基因流动的证据

Author Correction: Defects in mtDNA replication challenge nuclear genome stability through nucleotide depletion and provide a unifying mechanism for mouse progerias

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