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Synergy between an IGF-1R antibody and Raf/MEK/ERK and PI3K/Akt/mTOR pathway inhibitors in suppressing IGF-1R-mediated growth in hematopoietic cells

机译：IGF-1R抗体和RAF / MEK / ERK和PI3K / AKT / MTOR途径抑制剂在抑制IGF-1R介导的造血细胞生长之间的协同作用

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F E Bertrand; L S Steelman; W H Chappell; S L Abrams; J G Shelton; E R White; D L Ludwig; J A McCubrey;
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1. Synergy between an IGF-1R antibody and Raf|[sol]|MEK|[sol]|ERK and PI3K|[sol]|Akt|[sol]|mTOR pathway inhibitors in suppressing IGF-1R-mediated growth in hematopoietic cells [J] . F E Bertrand, L S Steelman, W H Chappell, Leukemia . 2006,第7期

机译：IGF-1R抗体与Raf | [sol] | MEK | [sol] | ERK和PI3K | [sol] | Akt | [sol] | mTOR途径抑制剂之间的协同作用可抑制IGF-1R介导的造血细胞生长
2. Synergy between PI3K/Akt and Raf/MEK/ERK pathways in IGF-1R mediated cell cycle progression and prevention of apoptosis in hematopoietic cells. [J] . Shelton JG, Steelman LS, White ER, Cell cycle . 2004,第3期

机译：IGF-1R中PI3K / Akt和Raf / MEK / ERK途径之间的协同作用介导了细胞周期进程并阻止了造血细胞的凋亡。
3. Synergy between PI3K/Akt and Raf/MEK/ERK pathways in IGF-1R mediated cell cycle progression and prevention of apoptosis in hematopoietic cells. [J] . Shelton JG, Steelman LS, White ER, Cell cycle . 2004,第3期

机译：IGF-1R中PI3K / Akt和Raf / MEK / ERK途径之间的协同作用介导了细胞周期进程并阻止了造血细胞的凋亡。
4. Combination of PI3K/Akt/mTOR inhibitors and PDT in endothelial and tumor cells. [C] . Fateye, Babasola, Chen Bin Optical methods for tumor treatment and detection: mechanisms and techniques in photodynamic therapy XX . 2011

机译：内皮细胞和肿瘤细胞中PI3K / Akt / mTOR抑制剂和PDT的组合。
5. Abrogation of cytokine dependency in hematopoietic cells by Raf and PI3K/Akt. [D] . Pohnert, Steven Clark. 2002

机译：Raf和PI3K / Akt消除造血细胞中细胞因子的依赖性。
6. P-021Tanshinone IIA can inhibit human pancreatic carcinoma BxPC3 cells through decreasing the protein expressions of IGFR EGFR VEGFR and blocking both Ras/Raf/MEK/ERK and Ras/PI3K/AKT/mTOR pathway in vitro and in vivo [O] . C.C. Su -1

机译：P-021丹参酮IIA可以通过降低IGFREGFRVEGFR的蛋白质表达并阻断Ras / Raf / MEK / ERK和Ras / PI3K / AKT / mTOR通路来抑制人胰腺癌BxPC3细胞
7. Tanshinone IIA can inhibit MiaPaCa‑2 human pancreatic cancer cells by dual blockade of the Ras/Raf/MEK/ERK and PI3K/AKT/mTOR pathways [O] . Chin‑Cheng Su 2018

机译：丹参酮IIa可以通过RAS / RAF / MEK / ERK和PI3K / AKT / MTOR途径的双封闭式抑制MIAPACA-2人胰腺癌细胞

Synergy between an IGF-1R antibody and Raf/MEK/ERK and PI3K/Akt/mTOR pathway inhibitors in suppressing IGF-1R-mediated growth in hematopoietic cells

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