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Translisin recognition site sequences flank translocation breakpoints in a Philadelphia chromosome positive chronic myeloid leukemia patient expressing a novel type of chimeric BCR-ABL transcript (E8-INT-A2)

机译：译民识别位点序列在费城染色体阳性慢性骨髓白血病患者中表达新型嵌合BCR-ABL转录物（E8-INT-A2）

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G Martinelli; C Terragna; M Amabile; V Montefusco; N Testoni; E Ottaviani; A de Vivo; A Mianulli; E Trabacchi; G Saglio; S Tura;
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1. Alu and translisin recognition site sequences flanking translocation sites in a novel type of chimeric bcr-abl transcript suggest a possible general mechanism for bcr-abl breakpoints | Haematologica [J] . G Martinelli, C Terragna, M Amabile, Haematologica . 2000,第1期

机译：新型的嵌合bcr-abl转录本中位于易位位点两侧的Alu和Translisin识别位点序列提示了bcr-abl断点的可能一般机制|血液学
2. Novel types of bcr-abl transcript with breakpoints in BCR exon 8 found in Philadelphia positive patients with typical chronic myeloid leukemia retain the sequence encoding for the DBL- and CDC24 homology domains but not the pleckstrin homology one | Haematologica [J] . G Martinelli, M Amabile, B Giannini, Haematologica . 2002,第7期

机译：在典型的慢性髓性白血病的费城阳性患者中发现的新型Bcr-abl转录物具有BCR外显子8的断裂点，保留了编码DBL-和CDC24同源结构域的序列，但不保留pleckstrin同源序列。血液学
3. Aberrant BCR-ABL transcript with intronic insertion in a patient with philadelphia chromosome-positive chronic myeloid leukemia: implications for disease progression. [J] . Shiratsuchi M, Muta K, Minami R, Leukemia and lymphoma . 2001,第3a4期

机译：费城染色体阳性慢性粒细胞白血病患者中内含子插入的异常BCR-ABL转录本：对疾病进展的影响。
4. A rare chronic myeloid leukemia case with Philadelphia chromosome BCR-ABL e13a3 transcript and complex translocation involving four different chromosomes [O] . WALID AL ACHKAR, ABDULSAMAD WAFA, BASHAR YOUSEF ALI, -1

机译：罕见的慢性粒细胞白血病病例有费城染色体BCR-ABL e13a3转录本和涉及四个不同染色体的复杂易位
5. Antisense inhibition of Bcr-Abl/c-Abl synthesis promotes telomerase activity and upregulates tankyrase in human leukemia cells11The GenBank accession numbers for human Bcr-Abl and human c-Abl cDNA sequences described in this paper are X02596/M24603 and X16416, respectively. ODN1/PS-ODN1 antisense sequences and ODN3/PS-ODN3 control sense sequences correspond to the nucleotide sites localized in the breakpoint cluster region (b3a2 junction) in the 9:22 chromosomal translocation (Philadelphia chromosome, accession numbers X02596 and M24603, version X02596.1, between ex. 3061 and 3121). ODN2/PS-ODN2 antisense sequences and ODN4/PS-ODN4 control sense sequences correspond to the nucleotide site of Abl proto-oncogene, localized between ex. 961 and 1021 (accession number X16416, version X16416.1). ODN5 and PS-ODN5 are nonsense sequences and did not show homology with both cDNAs. [O] . Bakalova Rumiana, Ohba Hideki, Zhelev Zhivko, 2004

机译：对Bcr-Abl / c-Abl合成的反义抑制可促进人白血病细胞中的端粒酶活性并上调tankase酶11本文描述的人Bcr-Abl和人c-Abl cDNA序列的GenBank登录号分别为X02596 / M24603和X16416。 ODN1 / PS-ODN1反义序列和ODN3 / PS-ODN3控制有义序列对应于9:22染色体易位（费城染色体，登录号X02596和M24603，版本X02596）中位于断点簇区域（b3a2连接）的核苷酸位点.1（例如3061和3121之间）。 ODN2 / PS-ODN2反义序列和ODN4 / PS-ODN4控制有义序列对应于Abl原癌基因的核苷酸位点，位于ex。 961和1021（登录号X16416，版本X16416.1）。 ODN5和PS-ODN5是无义序列，与两个cDNA没有同源性。

Translisin recognition site sequences flank translocation breakpoints in a Philadelphia chromosome positive chronic myeloid leukemia patient expressing a novel type of chimeric BCR-ABL transcript (E8-INT-A2)

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