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Studies on the Contribution of Human Cytomegalovirus UL21a and UL97 to Viral Growth and Inactivation of the Anaphase-Promoting Complex/Cyclosome (APC/C) E3 Ubiquitin Ligase Reveal a Unique Cellular Mechanism for Downmodulation of the APC/C Subunits APC1, APC4, and APC5

机译：人巨细胞病毒UL21A和UL97对促进复合物复合/环状体（APC / C）的病毒生长和灭活的研究揭示了APC / C亚基APC1，APC4和APC5的滴定调节的独特细胞机制

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Elizabeth Clark; Deborah H. Spector;
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1. Cdh1, a substrate-recruiting component of anaphase-promoting complex/cyclosome (APC/C) ubiquitin E3 ligase, specifically interacts with phosphatase and tensin homolog (PTEN) and promotes its removal from chromatin. [J] . Byeong Hyeok Choi, Michele Pagano, Chuanshu Huang, The Journal of biological chemistry . 2017,第48期

机译：Cdh1，后期促进复合物/环体（APC / C）泛素E3连接酶的底物招募成分，与磷酸酶和肌腱蛋白同源物（PTEN）特异性相互作用，并促进其从染色质中的去除。
2. Cdh1, a Substrate-recruiting Component of Anaphase-promoting Complex/Cyclosome (APC/C) Ubiquitin E3 Ligase, Specifically Interacts with Phosphatase and Tensin Homolog (PTEN) and Promotes Its Removal from Chromatin [J] . Byeong Hyeok Choi, Michele Pagano, Chaunshu Huang, The Journal of biological chemistry . 2014,第25期

机译：CDH1，促进复合体/环体（APC / C）泛素E3连接酶的底物募集组分，与磷酸酶和硫素同源物（PTEN）特异性相互作用，并促进其从染色质中的去除
3. The Apc5 Subunit of the Anaphase-Promoting Complex/Cyclosome Interacts with Poly(A) Binding Protein and Represses Internal Ribosome Entry Site-Mediated Translation [J] . Nadejda Koloteva-Levine, Dalia Pinchasi, Idan Pereman, Molecular and Cellular Biology . 2004,第9期

机译：Apc5亚基的后期促进复杂/ Cyclosome与Poly（A）结合蛋白相互作用，并抑制内部核糖体进入位点介导的翻译。
4. Studies on the Contribution of Human Cytomegalovirus UL21a and UL97 to Viral Growth and Inactivation of the Anaphase-Promoting Complex/Cyclosome (APC/C) E3 Ubiquitin Ligase Reveal a Unique Cellular Mechanism for Downmodulation of the APC/C Subunits APC1 APC4 and APC5 [O] . Elizabeth Clark, Deborah H. Spector 2015

机译：人类巨细胞病毒UL21a和UL97对病毒生长和后期促进复合物/循环体（APC / C）E3泛素连接酶失活的贡献研究揭示了独特的细胞机制用于下调APC / C亚基APC1APC4和APC5
5. Inactivation and Disassembly of the Anaphase-Promoting Complex during Human Cytomegalovirus Infection Is Associated with Degradation of the APC5 and APC4 Subunits and Does Not Require UL97-Mediated Phosphorylation of Cdh1 ▿ [O] . Tran, Karen, Kamil, Jeremy P., Coen, Donald M., 2010

机译：人巨细胞病毒感染过程中后期促进复合物的失活和解体与APC5和APC4亚基的降解有关，并且不需要UL97介导的Cdh1磷酸化。

Studies on the Contribution of Human Cytomegalovirus UL21a and UL97 to Viral Growth and Inactivation of the Anaphase-Promoting Complex/Cyclosome (APC/C) E3 Ubiquitin Ligase Reveal a Unique Cellular Mechanism for Downmodulation of the APC/C Subunits APC1, APC4, and APC5

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