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DEVELOPMENT OF NEUROLOGICAL DEFICIT AFTER PERINATAL HYPOXIA AND EARLY LIFE STRESS IN LABORATORY ANIMALS

机译：围产期缺氧和实验动物早期生命压力后神经缺陷的发展

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Yu.A. Uspenskaya; N.A. Malinovskaya; V.V. Volkova; Yu.A. Panina; R.V. Ryabokon; O.V. Frolova; A.B. Salmina;
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1. Chronic neurological deficits in mice after perinatal hypoxia and ischemia correlate with hemispheric tissue loss and white matter injury detected by MRI. [J] . Cengiz P, Uluc K, Kendigelen P, Developmental Neuroscience . 2011,第3a4期

机译：围产期缺氧和局部缺血后小鼠的慢性神经功能缺损与MRI检测到的半球组织丢失和白质损伤有关。
2. Developmental motor deficits induced by combined fetal exposure to lipopolysaccharide and early neonatal hypoxia/ischemia: a novel animal model for cerebral palsy in very premature infants. [J] . Girard S, Kadhim H, Beaudet N, Neuroscience: An International Journal under the Editorial Direction of IBRO . 2009,第2期

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4. Neurological Deficit Rather than Obliteration Determines Quality of Life in Patients Treated with Radiosurgery for AVMs [C] . Meera Ramani, Yuri M. Andrade-Souza, Deirdre R. Dawson, International Stereotactic Radiosurgery Society . 2006

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5. The Role of Fibroblast Growth Factors in Cortical Regeneration After Perinatal Hypoxia: A Model For Neurological Recovery In Premature Children. [D] . Fagel, Devon Marc. 2013

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6. Chronic Neurological Deficits in Mice after Perinatal Hypoxia and Ischemia Correlate with Hemispheric Tissue Loss and White Matter Injury Detected by MRI [O] . Pelin Cengiz, Kutluay Uluc, Pinar Kendigelen, -1

机译：围产期缺氧和缺血后小鼠的慢性神经系统缺陷与MRI检测到的半球组织丢失和白质损伤相关
7. Chronic Neurological Deficits in Mice after Perinatal Hypoxia and Ischemia Correlate with Hemispheric Tissue Loss and White Matter Injury Detected by MRI [O] . Pelin Cengiz, Kutluay Uluc, Pinar Kendigelen, 2011

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8. Workshop on the Qualitative and Quantitative Comparability of Human and Animal Developmental Neurotoxicity, Work Group 1 Report: Comparability of Measures of Developmental Neurotoxicity in Humans and Laboratory Animals [R] . Stanton, M. E., Spear, L. P. 1990

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DEVELOPMENT OF NEUROLOGICAL DEFICIT AFTER PERINATAL HYPOXIA AND EARLY LIFE STRESS IN LABORATORY ANIMALS

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