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P059. Human alpha-Defensin 6 regulated by both Atoh1 and beta-catenin might be the pathogenesis of Japanese Crohn's Disease

机译：P059。由atoh1和beta-catenin调节的人α-defensin 6可能是日本克罗恩病的发病机制

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1. Reduced Human alpha-defensin 6 in Noninflamed Jejunal Tissue of Patients with Crohn's Disease [J] . Hayashi Ryohei, Tsuchiya Kiichiro, Fukushima Keita, Inflammatory bowel diseases . 2016,第5期

机译：克罗恩病患者非发炎的空肠组织中人α-防御素6含量降低
2. Crohn's disease-specific pancreatic autoantibodies are specifically present in ruminants with paratuberculosis: Implications for the pathogenesis of the human disease [J] . LiaskosC., SpyrouV., RoggenbuckD., Autoimmunity . 2013,第6期

机译：克罗恩病特异性胰腺自身抗体特别存在于副结核病反刍动物中：对人类疾病发病机制的影响
3. Age-of-diagnosis dependent ileal immune intensification and reduced alpha-defensin in older versus younger pediatric Crohn Disease patients despite already established dysbiosis [J] . Haberman Yael, Schirmer Melanie, Dexheimer Phillip J., Mucosal immunology . 2019,第2期

机译：诊断年龄依赖性髂骨免疫增强和较年轻的儿科克罗恩病患者的α-defensin尽管已经建立了困难
4. Exclusive Enteral Nutrition: Clues to the Pathogenesis of Crohn's Disease [C] . Arie Levine Nestlé Nutrition Workshop . 2014

机译：独家肠内营养：克罗恩病发病机制的线索
5. Identifying new therapeutic targets for the treatment of Crohn's disease: The role of CD47 and L-carnitine in the pathogenesis and treatment of a murine model of intestinal inflammation. [D] . Fortin, Genevieve. 2010

机译：确定克罗恩病的新治疗靶标：CD47和L-肉碱在肠道炎症的鼠模型的发病机理和治疗中的作用。
6. Age-of-Diagnosis Dependent Ileal Immune Intensification and Reduced Alpha-Defensin in Older versus Younger Pediatric Crohn Disease Patients despite Already Established Dysbiosis [O] . Yael Haberman, Melanie Schirmer, Phillip J. Dexheimer, -1

机译：尽管已经建立了营养不良症但老年与年轻小儿克罗恩病患者的诊断依赖年龄的回肠免疫强度增强和α-防御素降低
7. Alpha-defensins (α-Defs) in Crohn's disease: decrease of ileal α-Def 5 via permanent methylation and increase in plasma α-Def 1-3 concentrations offering biomarker utility [O] . E. Cerrillo, I. Moret, M. Iborra, 2018

机译：Crohn疾病中的α-防御素（α-DEFS）：通过永久性甲基化的ILALα-DEF 5降低，血浆α-DEF 1-3浓度提供生物标志物效用

P059. Human alpha-Defensin 6 regulated by both Atoh1 and beta-catenin might be the pathogenesis of Japanese Crohn's Disease

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