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Discovery of a novel phosphoinositide 3-kinase gamma (PI3Kγ) inhibitor against hematologic malignancies and theoretical studies on its PI3Kγ-specific binding mechanisms

机译：发现新型磷酸阳性3-激酶γ（PI3Kγ）抑制作用于其PI3Kγ特异性结合机制的理论研究

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Jingyu Zhu; Ke Ke; Lei Xu; Jian Jin;
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1. Discovery of a novel phosphoinositide 3-kinase gamma (PI3K gamma) inhibitor against hematologic malignancies and theoretical studies on its PI3K gamma-specific binding mechanisms [J] . Zhu Jingyu, Ke Ke, Xu Lei, RSC Advances . 2019,第35期

机译：发现新型磷酸阳性3-激酶γ（PI3Kγ）抑制剂对血液学恶性肿瘤的理论研究及其PI3Kγ特异性结合机制
2. Discovery of a novel phosphoinositide 3-kinase gamma (PI3Kγ) inhibitor against hematologic malignancies and theoretical studies on its PI3Kγ-specific binding mechanisms [J] . Jingyu Zhu, Ke Ke, Lei Xu, RSC Advances . 2019,第35期

机译：新型抗血液恶性肿瘤的磷酸肌醇3-激酶γ（PI3Kγ）抑制剂的发现及其PI3Kγ特异性结合机制的理论研究
3. Discovery of a novel phosphoinositide 3-kinase gamma (PI3Kγ) inhibitor against hematologic malignancies and theoretical studies on its PI3Kγ-specific binding mechanisms [J] . Jingyu Zhu, Ke Ke, Lei Xu, RSC Advances . 2019,第35期

机译：新型抗血液恶性肿瘤的磷酸肌醇3-激酶γ（PI3Kγ）抑制剂的发现及其PI3Kγ特异性结合机制的理论研究
4. Inositol (1,4,5) trisphosphate 3-kinase B (ItpkB) is central to thymic development by regulating Phosphoinositide 3-kinase (PI3K) downstream of mild and strong TCR signals. [D] . Sternberg, Luise. 2013

机译：肌醇（1,4,5）三磷酸3激酶B（ItpkB）通过调节轻度和强TCR信号下游的磷酸肌醇3激酶（PI3K）对胸腺发育至关重要。
5. Opportunistic Infections (OIs) in Patients with Hematologic Malignancies (HM) Treated with Bruton’s Tyrosine Kinase (BTK) and Phosphoinositide 3 Kinase (PI3K) Inhibitors: An 8-Year Retrospective Cohort Study [O] . Nicolas Issa, Esther Arbona-Haddad, Alexandra Nevett-Fernandez, 2017

机译：布鲁顿酪氨酸激酶（BTK）和磷酸肌醇3激酶（PI3K）抑制剂治疗的血液系统恶性肿瘤（HM）患者的机会感染（OIs）：一项为期8年的回顾性队列研究
6. A Switch of G Protein-Coupled Receptor Binding Preference from Phosphoinositide 3-Kinase (PI3K)–p85 to Filamin A Negatively Controls the PI3K Pathway [O] . Najiba, Souad, Saint-Laurent, Nathalie, Estève, Jean-Pierre, 2012

机译：从磷酸肌醇3-激酶（PI3K）–p85到丝蛋白A的G蛋白偶联受体结合偏好的转换对PI3K通路产生负面影响。

Discovery of a novel phosphoinositide 3-kinase gamma (PI3Kγ) inhibitor against hematologic malignancies and theoretical studies on its PI3Kγ-specific binding mechanisms

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