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Preferential induction of G1 arrest in androgen-responsive human prostate cancer cells by androgen receptor signaling antagonists DL3 and antiandrogen bicalutamide

机译：雄激素受体信号传导拮抗剂DL3和抗抗原酰胺癌细胞对雄激素响应人前列腺癌细胞的优先诱导G1停滞

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Shan Lu; Zongqin Tan; Matthew Wortman; Zhongyun Dong;
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1. Preferential induction of G1 arrest in androgen-responsive human prostate cancer cells by androgen receptor signaling antagonists DL3 and antiandrogen bicalutamide. [J] . Tan Z, Wortman M, Lu S, Cancer letters . 2010,第2期

机译：雄激素受体信号传导拮抗剂DL3和抗雄激素比卡鲁胺可优先诱导雄激素反应性人前列腺癌细胞中G1阻滞。
2. The antiandrogen bicalutamide activates the androgen receptor (AR) with a mutation in codon 741 through the mitogen activated protein kinase (MARK) pathway in human prostate cancer PC3 cells. [J] . Terakawa T, Miyake H, Kumano M, Oncology reports . 2010,第5期

机译：抗雄激素比卡鲁胺通过人前列腺癌PC3细胞中的有丝分裂原活化蛋白激酶（MARK）途径激活741密码子突变的雄激素受体（AR）。
3. 17beta-Estradiol differentially regulates androgen-responsive genes through estrogen receptor-beta- and extracellular-signal regulated kinase-dependent pathways in LNCaP human prostate cancer cells. [J] . Takahashi Y, Perkins SN, Hursting SD, Molecular Carcinogenesis . 2007,第2期

机译：17β-雌二醇通过LNCaP人前列腺癌细胞中的雌激素受体-β和细胞外信号调节的激酶依赖性途径，差异调节雄激素应答基因。
4. Proteome-wide drug dose-response of prostate cancer cell lines exposed to androgen receptor antagonist by microflow-LC SWATH MS analysis [C] . L.C. Gillet, S. Amon, Y. Uchida, ASMS Conference on Mass Spectrometry and Allied Topics . 2016

机译：通过Microflow-LC SWATH MS分析将前列腺癌细胞拮抗剂暴露于雄激素受体拮抗剂的前列腺癌细胞响应
5. Development of pan-antagonists of mutant androgen receptors associated with antiandrogen resistant prostate cancer. [D] . Pan, Hongmu. 2009

机译：与抗雄激素抵抗性前列腺癌有关的突变雄激素受体泛拮抗剂的发展。
6. Preferential Induction of G1 Arrest in Androgen Responsive Human Prostate Cancer Cells by Androgen Receptor Signaling Antagonists DL3 and antiandrogen bicalutamide [O] . Shan Lu, Zongqin Tan, Matthew Wortman, -1

机译：雄激素受体信号传导拮抗剂DL3和抗抗原与丁酰胺雄激素响应人前列腺癌细胞优先诱导雄激素响应人前列腺癌细胞
7. Galbanic acid decreases androgen receptor abundance and signaling and induces G1 arrest in prostate cancer cells [O] . Yong Zhang, Kwan-Hyun Kim, Wei Zhang, 2011

机译：Galbanic acid降低雄激素受体丰度和信号传导，并在前列腺癌细胞中诱导G1停滞
8. Molecular Determinants of Prostate Cancer Progression Across Race- Ethnicity. Project A - The Human 5RD5A2 Gene and Prostate Cancer Progression. Project B - Androgen Receptor (AR) Signaling in Prostate Cancer Progression. Project C - Cellular and Molecular Markers of Prostate Cancer Progression Core - Epidemiology Core [R] . Ross, R. R. , Reichardt, J. , Coetzee, G. A. , 2002

机译：跨越种族的前列腺癌进展的分子决定因素。项目a - 人类5RD5a2基因和前列腺癌进展。项目B - 雄激素受体（aR）在前列腺癌进展中的信号传导。项目C - 前列腺癌进展核心的细胞和分子标记 - 流行病学核心

Preferential induction of G1 arrest in androgen-responsive human prostate cancer cells by androgen receptor signaling antagonists DL3 and antiandrogen bicalutamide

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