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Ligand-Dependent Activation of the Chimeric Tumor Necrosis Factor Receptor-Amyloid Precursor Protein (APP) Reveals Increased APP Processing and Suppressed Neuronal Differentiation

机译：嵌合肿瘤坏死因子受体 - 淀粉样蛋白前体蛋白（APP）的配体依赖性活化揭示了增加的应用加工和抑制神经元分化

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Poh-Ling Sim; Klaus Heese;
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1. Ligand-Dependent Activation of the Chimeric Tumor Necrosis Factor Receptor-Amyloid Precursor Protein (APP) Reveals Increased APP Processing and Suppressed Neuronal Differentiation [J] . Poh-Ling Sim, Klaus Heese Neuro-signals . 2010,第1期

机译：嵌合肿瘤坏死因子受体淀粉样前体蛋白（APP）的配体依赖性激活显示增加的APP处理和抑制的神经元分化。
2. Ligand-Dependent Activation of the Chimeric Tumor Necrosis Factor Receptor-Amyloid Precursor Protein (APP) Reveals Increased APP Processing and Suppressed Neuronal Differentiation [J] . Poh-Ling Sim, Klaus Heese Neuro-signals . 2010,第1期

机译：嵌合肿瘤坏死因子受体淀粉样前体蛋白（APP）的配体依赖性激活显示增加的APP处理和抑制的神经元分化。
3. Semi-vioxanthin Isolated from Marine-Derived Fungus Regulates Tumor Necrosis Factor-alpha, Cluster of Differentiation (CD) 80, CD86, and Major Histocompatibility Complex Class II Expression in RAW264.7 Cells via Nuclear Factor-kappa B and Mitogen-Activated Protein Kinase Signaling Pathways [J] . Yang XY, Cai SX, Zhang WJ, Biological & pharmaceutical bulletin . 2008,第12期

机译：从海洋衍生真菌中分离出的半紫黄质通过核因子-κB和丝裂原活化的蛋白激酶调节肿瘤坏死因子-α，分化簇（CD）80，CD86和主要组织相容性复合物II类在RAW264.7细胞中的表达。信号通路
4. Pravastatin and Rosiglitazone May Attenuate Coronary Artery Disease by Decreasing Interferon-gamma, Tumor Necrosis Factor-alpha, Interleukin-6, Macrophage Chemotactic Protein-1, C-Reactive Protein, and Increasing Interleukin-4 [C] . L.G. Tan, S.A. Tan, L.S. Berk International Congress on Coronary Artery Disease . 2007

机译：普伐他汀和罗格列酮可以通过减少干扰素-γ，肿瘤坏死因子-α，白细胞介素-6，巨噬细胞趋化蛋白-1，C反应蛋白和增加白细胞介素-4
5. The regulation of the monocyte chemoattractant protein-1 by tumor necrosis factor-alpha requires activation of transcription factors, chromatin modification, and long distance protein-protein communication. [D] . Boekhoudt, Gunther Heinrich. 2003

机译：肿瘤坏死因子-α对单核细胞趋化蛋白-1的调节需要激活转录因子，染色质修饰和长距离蛋白-蛋白通讯。
6. Chimeric tumor necrosis factor-TrkA receptors reveal that ligand-dependent activation of the TrkA tyrosine kinase is sufficient for differentiation and survival of PC12 cells. [O] . G Rovelli, R A Heller, M Canossa, 1993

机译：嵌合肿瘤坏死因子-TrkA受体表明TrkA酪氨酸激酶的配体依赖性激活足以使PC12细胞分化和存活。
7. Chimeric tumor necrosis factor-TrkA receptors reveal that ligand-dependent activation of the TrkA tyrosine kinase is sufficient for differentiation and survival of PC12 cells. [O] . Rovelli, G, Heller, R A, Canossa, M, 1993

机译：嵌合肿瘤坏死因子-TrkA受体表明，TrkA酪氨酸激酶的配体依赖性激活足以使PC12细胞分化和存活。
8. Hantaan Virus Nucleocapsid Protein Binds to Importin alpha Proteins and Inhibits Tumor Necrosis Factor Alpha-Induced Activation of Nuclear Factor Kappa B [R] . Taylor, S. L., Frias-Staheli, N., Garcia-Sastre, A., 2008

机译：汉坦病毒核衣壳蛋白与进口蛋白α蛋白结合并抑制肿瘤坏死因子α诱导的核因子κB活化

Ligand-Dependent Activation of the Chimeric Tumor Necrosis Factor Receptor-Amyloid Precursor Protein (APP) Reveals Increased APP Processing and Suppressed Neuronal Differentiation

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