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Study of an extended family with CTLA-4 deficiency suggests a CD28/CTLA-4 independent mechanism responsible for differences in disease manifestations and severity

机译：具有CTLA-4缺乏的大家庭的研究表明CD28 / CTLA-4独立机制，负责疾病表现和严重程度的差异

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Tie Zheng Hou; Peter Olbrich; Jose Manuel Lucena Soto; Berta Sanchez; Paula Sanchez Moreno; Stephan Borte; Hans J. Stauss; Siobhan O. Burns; Lucy S.K. Walker; Qiang Pan-Hammarström; Lennart Hammarström; David M. Sansom; Olaf Neth;
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1. Study of an extended family with CTLA-4 deficiency suggests a CD28/CTLA-4 independent mechanism responsible for differences in disease manifestations and severity [J] . Hou Tie Zheng, Olbrich Peter, Lucena Soto Jose Manuel, Clinical immunology: The official journal of the Clinical Immunology Society . 2018,第期

机译：具有CTLA-4缺乏的大家庭的研究表明CD28 / CTLA-4独立机制，负责疾病表现和严重程度的差异
2. Dysregulated expression of both the costimulatory CD28 and inhibitory CTLA-4 molecules in PB T cells of advanced cervical cancer patients suggests systemic immunosuppression related to disease progression. [J] . Agata Kosmaczewska, Dorota Bocko, Lidia Ciszak, Pathology oncology research: POR . 2012,第2期

机译：在晚期宫颈癌患者的PB T细胞中，共刺激性CD28和抑制性CTLA-4分子的表达失调提示与疾病进展相关的全身性免疫抑制。
3. Plasma concentrations of soluble CTLA-4, CD28, CD80 and CD86 costimulatory molecules reflect disease severity of acute asthma in children. [J] . Ip WK, Wong CK, Leung TF, Pediatric Pulmonology . 2006,第7期

机译：可溶性CTLA-4，CD28，CD80和CD86共刺激分子的血浆浓度反映了儿童急性哮喘的疾病严重程度。
4. Dysregulated Expression of Both the Costimulatory CD28 and Inhibitory CTLA-4 Molecules in PB T Cells of Advanced Cervical Cancer Patients Suggests Systemic Immunosuppression Related to Disease Progression [O] . Agata Kosmaczewska, Dorota Bocko, Lidia Ciszak, -1

机译：共刺激性CD28和抑制性CTLA-4分子在晚期宫颈癌患者PB T细胞中的表达失调提示与疾病进展相关的全身免疫抑制
5. Dysregulated Expression of Both the Costimulatory CD28 and Inhibitory CTLA-4 Molecules in PB T Cells of Advanced Cervical Cancer Patients Suggests Systemic Immunosuppression Related to Disease Progression [O] . Agata Kosmaczewska, Dorota Bocko, Lidia Ciszak, 2011

机译：共刺激性CD28和抑制性CTLA-4分子在晚期宫颈癌患者PB T细胞中的表达失调提示与疾病进展相关的全身免疫抑制

Study of an extended family with CTLA-4 deficiency suggests a CD28/CTLA-4 independent mechanism responsible for differences in disease manifestations and severity

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