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Oncogenic miR-20b-5p contributes to malignant behaviors of breast cancer stem cells by bidirectionally regulating CCND1 and E2F1

机译：通过双向调节CCND1和E2F1对乳腺癌干细胞的恶性行为有助于恶性乳腺癌干细胞的恶性行为

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Liqin Xia; Feng Li; Jun Qiu; Zhongming Feng; Zihan Xu; Zhengtang Chen; Jian-Guo Sun;
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1. LINC01087 is Highly Expressed in Breast Cancer and Regulates the Malignant Behavior of Cancer Cells Through miR-335-5p/Rock1 [J] . Ji-Kai She, Dan-Ni Fu, Dong Zhen, OncoTargets and therapy . 2020,第2期

机译：LINC01087在乳腺癌中高度表达，并通过MIR-335-5P / ROCK1调节癌细胞的恶性行为
2. Calcium sensing receptor down-regulates malignant cell behavior and promotes chemosensitivity in human breast cancer cells. [J] . Liu G, Hu X, Chakrabarty S Cell Calcium: The International Interdisciplinary Forum for Research on Calcium . 2009,第3期

机译：钙敏感受体下调人类乳腺癌细胞的恶性行为并促进其化学敏感性。
3. Developmental signaling pathways regulating mammary stem cells and contributing to the etiology of triple-negative breast cancer [J] . Rangel Maria Cristina, Bertolette Daniel, Castro Nadia P., Breast cancer research and treatment. . 2016,第2期

机译：调节乳干细胞并促进三阴性乳腺癌的病因的发育信号通路
4. Breast tumor cell behaviors on in vitro models mimicking extracellular matrix at different malignant stages [C] . Takashi Hoshiba, Masaru Tanaka Society for Biomaterials annual meeting and exposition . 2014

机译：模拟不同恶性阶段细胞外基质的体外模型上的乳腺癌细胞行为
5. Autocrine and Paracrine Signaling Regulates Breast Cancer Stem Cells and Epithelial-Mesenchymal Transition in Inflammatory Breast Cancer [D] . Valeta, Amanda. 2016

机译：自分泌和旁碱基信号调节炎症乳腺癌中的乳腺癌干细胞和上皮 - 间充质转换
6. Oncogenic miR-20b-5p contributes to malignant behaviors of breast cancer stem cells by bidirectionally regulating CCND1 and E2F1 [O] . Liqin Xia, Feng Li, Jun Qiu, 2020

机译：通过双向调节CCND1和E2F1对乳腺癌干细胞的恶性行为有助于恶性乳腺癌干细胞的恶性行为
7. Oncogenic miR-20b-5p contributes to malignant behaviors of breast cancer stem cells by bidirectionally regulating CCND1 and E2F1 [O] . Liqin Xia, Feng Li, Jun Qiu, 2020

机译：通过双向调节CCND1和E2F1对乳腺癌干细胞的恶性行为有助于恶性乳腺癌干细胞的恶性行为

Oncogenic miR-20b-5p contributes to malignant behaviors of breast cancer stem cells by bidirectionally regulating CCND1 and E2F1

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