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Gut Microbiota Dysbiosis Accelerates Prostate Cancer Progression Through Increased LPCAT1 Expression and Enhanced DNA Repair Pathways

机译：肠道微生物瘤功能亢进通过增加的LPCAT1表达和增强的DNA修复途径加速前列腺癌进展

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Yufei Liu; Chen Yang; Zheyu Zhang; Haowen Jiang;
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1. LPCAT1 enhances castration resistant prostate cancer progression via increased mRNA synthesis and PAF production [J] . Chao Han, Guopeng Yu, Yuanshen Mao, PLoS One . 2020,第11期

机译：LPCAT1通过增加的mRNA合成和PAF生产增强抗阉割前列腺癌进展
2. The expression level of lysophosphatidylcholine acyltransferase 1 (LPCAT1) correlates to the progression of prostate cancer [J] . ZhouX., LawrenceT.J., HeZ., Experimental & Molecular Pathology . 2012,第1期

机译：溶血磷脂酰胆碱酰基转移酶1（LPCAT1）的表达水平与前列腺癌的进展相关
3. Disorder of gut amino acids metabolism during CKD progression is related with gut microbiota dysbiosis and metagenome change [J] . Yang Liu, Jianping Li, Jingao Yu, Journal of Pharmaceutical and Biomedical Analysis: An International Journal on All Drug-Related Topics in Pharmaceutical, Biomedical and Clinical Analysis . 2018,第期

机译：CKD进展过程中肠道氨基酸代谢的紊乱与肠道微生物症失育病和Metagenome变化有关
4. Gene expression and pathway bioinformatics analysis detect a potential predictive value of MAP3K8 in thyroid cancer progression [C] . Valentina Di Salvatore, Fiorenza Gianì, Giulia Russo, IEEE International Conference on Bioinformatics and Biomedicine . 2019

机译：基因表达和途径生物信息学分析检测MAP3K8在甲状腺癌进展中的潜在预测价值
5. Development and application of DNA damage and DNA repair capacity indices to prostate cancer. [D] . Nock, Nora Lynne. 2005

机译：DNA损伤和DNA修复能力指标在前列腺癌中的开发和应用。
6. Reduced stress-associated FKBP5 DNA methylation together with gut microbiota dysbiosis is linked with the progression of obese PCOS patients [O] . Fu Chen, Zhangran Chen, Minjie Chen, 2021

机译：减少应力相关的FKBP5 DNA甲基化与肠道微生物症脱虫病与肥胖PCOS患者的进展相关联
7. LPCAT1 enhances castration resistant prostate cancer progression via increased mRNA synthesis and PAF production [O] . Chao Han, Guopeng Yu, Yuanshen Mao, 2020

机译：LPCAT1通过增加的mRNA合成和PAF生产增强抵抗前列腺癌进展

Gut Microbiota Dysbiosis Accelerates Prostate Cancer Progression Through Increased LPCAT1 Expression and Enhanced DNA Repair Pathways

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