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Opioid growth factor and low-dose naltrexone impair central nervous system infiltration by CD4 + T lymphocytes in established experimental autoimmune encephalomyelitis, a model of multiple sclerosis

机译：Apioid生长因子和低剂量纳曲酮受到CD4 + T淋巴细胞在已建立的实验性自身免疫性脑脊髓炎中的CD4 + T淋巴细胞的损伤中枢神经系统浸润，一种多发性硬化模型

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Leslie A Hammer; Hanspeter Waldner; Ian S Zagon; Patricia J McLaughlin;
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1. Opioid growth factor and low-dose naltrexone impair central nervous system infiltration by CD4+T lymphocytes in established experimental autoimmune encephalomyelitis, a model of multiple sclerosis [J] . Hammer Leslie A., Waldner Hanspeter, Zagon Ian S., Experimental Biology and Medicine: Journal of the Society for Experimental Biology and Medicine . 2016 ,第1期

机译：阿片类生长因子和低剂量纳曲酮通过建立的实验性自身免疫性脑脊髓炎（一种多发性硬化症模型）损害CD4 + T淋巴细胞对中枢神经系统的浸润
2. Prevention and diminished expression of experimental autoimmune encephalomyelitis by low dose naltrexone (LDN) or opioid growth factor (OGF) for an extended period: Therapeutic implications for multiple sclerosis [J] . Kristen A. Rahn, Patricia J. McLaughlin, Ian S. Zagon Brain research . 2011 ,第Null期

机译：低剂量纳曲酮（LDN）或阿片类生长因子（OGF）长期预防和减少实验性自身免疫性脑脊髓炎的表达：对多发性硬化症的治疗意义
3. The Ifng gene is essential for Vdr gene expression and vitamin D 3-mediated reduction of the pathogenic T cell burden in the central nervous system in experimental autoimmune encephalomyelitis, a multiple sclerosis model [J] . SpanierJ.A., NasholdF.E., OlsonJ.K., The Journal of Immunology: Official Journal of the American Association of Immunologists . 2012 ,第6期

机译：在实验性自身免疫性脑脊髓炎（一种多发性硬化模型）中，Ifng基因对于Vdr基因表达和维生素D 3介导的中枢神经系统病原性T细胞负担的减少至关重要。
4. Nanomaterials for immunotherapy against multiple sclerosis/experimental autoimmune encephalomyelitis (EAE) [C] . Alireza Hassani Najafabadi, Rui Kuai, Anna Schwendeman, Society for Biomaterials annual meeting and exposition . 2018

机译：纳米材料用于多发性硬化/实验性自身免疫性脑脊髓炎（EAE）的免疫治疗
5. STAT4 regulates multiple pathogenic mechanisms of CD4 T cells in experimental autoimmune encephalomyelitis, a model of multiple sclerosis [D] . McWilliams, Ian Lee 2015

机译：STAT4调节实验性自身免疫性脑脊髓炎（一种多发性硬化症的模型）中CD4 T细胞的多种致病机制
6. Opioid growth factor and low-dose naltrexone impair central nervous system infiltration by CD4 + T lymphocytes in established experimental autoimmune encephalomyelitis a model of multiple sclerosis [O] . Leslie A Hammer, Hanspeter Waldner, Ian S Zagon, 2016

机译：阿片样物质生长因子和低剂量纳曲酮通过建立的实验性自身免疫性脑脊髓炎（一种多发性硬化症模型）损害了CD4 + T淋巴细胞对中枢神经系统的浸润
7. Central nervous system infusion of TrkB agonist, 7,8-dihydroxyflavone, is ineffective in promoting myelin repair in cuprizone and experimental autoimmune encephalomyelitis mouse models of multiple sclerosis [O] . Jessica L Fletcher, Rhiannon J Wood, Alexa R Prawdiuk, 2021

机译：TRKB激动剂的中枢神经系统输注7,8-二羟基噻吩，在促进铜齐酮和实验性自身免疫性脑髓炎小鼠模型中促进髓鞘修复的无效性无效多发性硬化症
8. Isolating Selected Systems of Central Nervous System Tissue in Culture to Establish Models of Heat and Other Insult Stress-Prevention: Neurological Mechanisms of Heat Injury and Death [R] . Pleet, A. B., Fishman, R. H., Melamed, E., 1987

机译：在培养中分离中枢神经系统组织的选定系统以建立热和其他侮辱应激预防模型：热损伤和死亡的神经机制

Opioid growth factor and low-dose naltrexone impair central nervous system infiltration by CD4 + T lymphocytes in established experimental autoimmune encephalomyelitis, a model of multiple sclerosis

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