首页> 外文OA文献 >Deficits in synaptic function occur at medial perforant path-dentate granule cell synapses prior to Schaffer collateral-CA1 pyramidal cell synapses in the novel TgF344-Alzheimer's Disease Rat Model

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Deficits in synaptic function occur at medial perforant path-dentate granule cell synapses prior to Schaffer collateral-CA1 pyramidal cell synapses in the novel TgF344-Alzheimer's Disease Rat Model

机译：在新型TGF344-Alzheimer疾病大鼠疾病大鼠模型中，突触函数突触函数中的突触函数缺陷发生在中介穿孔路径牙齿颗粒细胞突触

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Lindsey A. Smith; Lori L. McMahon;
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1. Corrigendum to “Deficits in synaptic function occur at medial perforant path-dentate granule cell synapses prior to Schaffer collateral-CA1 pyramidal cell synapses in the novel TgF344-Alzheimer's Disease Rat Model” [Neurobiology of Disease Volume 110, February 2018, Pages 166–179] [J] . Lindsey A. Smith, Lori L. McMahon Neurobiology of disease . 2018,第期

机译：在新型TGF344-Alzheimer疾病大鼠模型中Schaffer壳体-CA1金字塔模型之前，“突触函数缺陷发生的突触函数缺陷发生的静脉曲张术”[2018年2月的疾病1月110日，第166-179页 ]
2. Deficits in synaptic function occur at medial perforant path-dentate granule cell synapses prior to Schaffer collateral-CA1 pyramidal cell synapses in the novel TgF344-Alzheimer's Disease Rat Model [J] . Lindsey A. Smith, Lori L. McMahon Neurobiology of disease . 2018,第期

机译：在新型TGF344-Alzheimer疾病大鼠大鼠模型中，在Schaffer Collateral-Ca1金字塔蛋白突触之前发生突触功能的缺陷发生在内侧穿孔路径牙齿颗粒细胞突触
3. Impaired short-term plasticity in mossy fiber synapses caused by mitochondrial dysfunction of dentate granule cells is the earliest synaptic deficit in a mouse model of Alzheimer's disease [J] . LeeS.H., KimK.-R., RyuS.-Y., The Journal of Neuroscience: The Official Journal of the Society for Neuroscience . 2012,第17期

机译：齿状颗粒细胞的线粒体功能障碍导致苔藓纤维突触的短期可塑性受损是阿尔茨海默氏病小鼠模型中最早的突触缺陷
4. Experimental validation of a synapse model by adding synaptic conductances to excitable endocrine cells in culture [C] . S. Boussa, M. Marin, F. LeFoll, European Symposium on Artificial Neural Networks . 2005

机译：通过向文化中添加突触导电来突触模型对培养中的激发内分泌细胞的实验验证
5. The Integration of Multiple Sources of Sonic Hedgehog in the Spinal Cord Contribute to the Production of First-Born Oligodendrocyte Precursor Cells Which Become Critical For Synapse Remodeling in Response to Adult Motor Neuron Injury [D] . Starikov, Lev. 2019

机译：脊髓中的脊髓刺猬的多种来源的整合有助于生产第一出生的寡核细胞前体细胞，这对于成年运动神经元损伤响应于突触重塑而变得至关重要
6. Deficits in synaptic function occur at medial perforant path-granule cell synapses prior to Schaffer collateral-CA1 pyramidal cell synapses in the novel TgF344-Alzheimer’s Disease Rat Model [O] . Lindsey A. Smith, Lori L. McMahon -1

机译：在新型TgF344-阿尔茨海默氏病大鼠模型中先于Schaffer侧支CA1锥体细胞突触的先于内侧穿孔路径-颗粒细胞突触发生突触功能的缺陷
7. Increased long-term potentiation at medial-perforant path-dentate granule cell synapses induced by selective inhibition of histone deacetylase 3 requires Fragile X mental retardation protein [O] . Aimee V. Franklin, James R. Rusche, Lori L. McMahon 2014

机译：通过选择性抑制组蛋白脱乙酰酶3的选择性抑制诱导的内侧穿孔路径牙齿颗粒细胞突触增加了长期增强需要脆弱的X精神迟滞蛋白
8. Osmolality-Induced Changes in Extracellular Volume Alter Epileptiform BurstsIndependent of Chemical Synapses in the Rat: Importance of Non-Synaptic Mechanisms in Hippocampal Epileptogenesis [R] . Dudek, F. E., Obenaus, A., Tasker, J. G. 1990

机译：渗透压诱导的细胞外体积变化改变癫痫样突发与大鼠化学突触无关：非突触机制在海马癫痫发生中的重要性

Deficits in synaptic function occur at medial perforant path-dentate granule cell synapses prior to Schaffer collateral-CA1 pyramidal cell synapses in the novel TgF344-Alzheimer's Disease Rat Model

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