首页> 外文OA文献 >Hrd1 and ER-Associated Protein Degradation, ERAD, Are Critical Elements of the Adaptive ER Stress Response in Cardiac Myocytes

【2h】

Hrd1 and ER-Associated Protein Degradation, ERAD, Are Critical Elements of the Adaptive ER Stress Response in Cardiac Myocytes

机译：HRD1和ER相关的蛋白质降解，ERAD是心肌细胞的适应性ER应激反应的关键要素

代理获取

本网站仅为用户提供外文OA文献查询和代理获取服务，本网站没有原文。下单后我们将采用程序或人工为您竭诚获取高质量的原文，但由于OA文献来源多样且变更频繁，仍可能出现获取不到、文献不完整或与标题不符等情况，如果获取不到我们将提供退款服务。请知悉。

页面导航

著录项
引文网络
相似文献
相关主题

著录项

作者
Shirin Doroudgar; Mirko Völkers; Donna J. Thuerauf; Mohsin Khan; Sadia Mohsin; Jonathan L. Respress; Wei Wang; Natalie Gude; Oliver J. Müller; Xander H.T. Wehrens; Mark A. Sussman; Christopher C. Glembotski;
展开▼
作者单位

展开▼
年度 2015
总页数
原文格式 PDF
正文语种
中图分类

相似文献

外文文献
中文文献
专利

1. Hrd1 and ER-Associated Protein Degradation, ERAD, Are Critical Elements of the Adaptive ER Stress Response in Cardiac Myocytes [J] . Doroudgar Shirin, Voelkers Mirko, Thuerauf Donna J., Circulation research: a journal of the American Heart Association . 2015,第6期

机译：Hrd1和ER相关蛋白降解，ERAD，是心肌细胞自适应ER应激反应的关键因素
2. Autoadaptive ER-Associated Degradation Defines a Preemptive Unfolded Protein Response Pathway (Retraction of vol 52, pg 783, 2013) [J] . Bernasconi Riccardo, Galli Carmela, Kokame Koichi, Molecular cell . 2014,第6期

机译：自适应ER相关降解定义了先发性未折叠的蛋白质反应途径（第52卷的撤回，第783页，2013年）
3. Autoadaptive ER-Associated degradation defines a preemptive unfolded protein response pathway [J] . BernasconiR., GalliC., KokameK., Molecular cell . 2013,第6期

机译：自适应ER相关降解定义了先发性未折叠蛋白反应途径
4. TOWARDS COMPUTATIONAL MODELING OF EXCITATION-CONTRACTION COUPLING IN CARDIAC MYOCYTES: RECONSTRUCTION OF STRUCTURES AND PROTEINS FROM CONFOCAL IMAGING [C] . FRANK B. SACHSE, ELEONORA SAVIO-GALIMBERTI, JOSHUA I. GOLDHABER, PSB;Pacific symposium on biocomputing; 20090105-09;20090105-09; Kohala Coast, HI(US);Kohala Coast, HI(US) . 2009

机译：心肌细胞兴奋-收缩耦合计算模型的研究：共聚焦成像重建结构和蛋白质
5. The physiology of mammalian ER quality control: Unfolded protein response and ER-associated degradation [D] . Yang, Liu. 2014

机译：哺乳动物ER质量控制的生理学：展开蛋白质反应和ER相关的降解
6. Hrd1 and ER-Associated Protein Degradation ERAD Are Critical Elements of the Adaptive ER Stress Response in Cardiac Myocytes [O] . Shirin Doroudgar, Mirko Völkers, Donna J Thuerauf, -1

机译：Hrd1和ER相关的蛋白降解ERAD是心肌细胞中自适应ER应激反应的关键因素
7. Protein disulfide isomerase-associated 6 is an ATF6-inducible ER stress response protein that protects cardiac myocytes from ischemia/reperfusion-mediated cell death [O] . John A. Vekich, Peter J. Belmont, Donna J. Thuerauf, 2012

机译：蛋白二硫化物异构酶相关的6是ATF6-诱导的ER应激反应蛋白，可保护心肌细胞免受缺血/再灌注介导的细胞死亡

Hrd1 and ER-Associated Protein Degradation, ERAD, Are Critical Elements of the Adaptive ER Stress Response in Cardiac Myocytes

著录项

引文网络

相似文献

相关主题

期刊订阅