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Elevated MeCP2 in Mice Causes Neurodegeneration Involving Tau Dysregulation and Excitotoxicity: Implications for the Understanding and Treatment of MeCP2 Triplication Syndrome

机译：小鼠的MECP2升高会导致涉及TAU失呼和兴奋毒性的神经变性：对MECP2三份综合征的理解和治疗的影响

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Kristen R. Montgomery; A. S. C. Louis Sam Titus; Lulu Wang; Santosh R. D’Mello;
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1. Elevated MeCP2 in Mice Causes Neurodegeneration Involving Tau Dysregulation and Excitotoxicity: Implications for the Understanding and Treatment of MeCP2 Triplication Syndrome [J] . Montgomery Kristen R., Titus A. S. C. Louis Sam, Wang Lulu, Molecular Neurobiology . 2018,第12期

机译：小鼠的MECP2升高导致涉及TAU失呼和兴奋毒性的神经变性：对MECP2三份综合征的理解和治疗的影响
2. Overexpression of methyl-CpG-binding protein 2 and autoimmunity: Evidence from MECP2 duplication syndrome, lupus, MECP2 transgenic and Mecp2 deficient mice [J] . SawalhaA.H. Lupus . 2013,第9期

机译：甲基CpG结合蛋白2的过表达和自身免疫：来自MECP2复制综合征，狼疮，MECP2转基因和Mecp2缺陷小鼠的证据
3. Selective preservation of cholinergic MeCP2 rescues specific Rett-syndrome-like phenotypes in MeCP2(stop) mice [J] . Zhou Huanhuan, Wu Wei, Zhang Ying, Behavioural Brain Research: An International Journal . 2017,第Pta1期

机译：Cholinergic MeCP2的选择性保存在MECP2（止动）小鼠中拯救了特定的RETT-综合征样表型
4. Inhibition of protein kinase C restores morphine efficacy following excitotoxic spinal cord injury: Implications for the treatment of post-spinal cord injury pain syndromes. [D] . Nolan, Todd Andrew. 2008

机译：抑制蛋白激酶C在兴奋性脊髓毒性损伤后恢复吗啡功效：对脊髓后损伤疼痛综合征的治疗意义。
5. Elevated MeCP2 in Mice Causes Neurodegeneration Involving Tau Dysregulation and Excitotoxicity: Implications for the Understanding and Treatment of MeCP2 Triplication Syndrome [O] . Kristen R. Montgomery, A. S. C. Louis Sam Titus, Lulu Wang, -1

机译：小鼠体内MeCP2升高导致涉及Tau失调和兴奋性毒性的神经退行性变：理解和治疗MeCP2三联综合征的意义。
6. Rett-causing mutations reveal two domains critical for MeCP2 function and for toxicity in MECP2 duplication syndrome mice [O] . Laura Dean Heckman, Maria H Chahrour, Huda Y Zoghbi 2014

机译：Rett导致突变显示了对MECP2功能和MECP2复制综合征小鼠毒性至关重要的两个结构域

Elevated MeCP2 in Mice Causes Neurodegeneration Involving Tau Dysregulation and Excitotoxicity: Implications for the Understanding and Treatment of MeCP2 Triplication Syndrome

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