首页> 外文OA文献 >Engagement of Adhesion Molecules (CD18, CD11a, CD45, CD44, and CD58) Enhances Human Immunodeficiency Virus Type 1 Replication in Monocytic Cells through a Tumor Necrosis Factor-Modulated Pathway

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Engagement of Adhesion Molecules (CD18, CD11a, CD45, CD44, and CD58) Enhances Human Immunodeficiency Virus Type 1 Replication in Monocytic Cells through a Tumor Necrosis Factor-Modulated Pathway

机译：粘附分子（CD18，CD11a，CD45，CD44和CD58）通过肿瘤坏死因子调制途径增强人免疫缺陷病毒1型复制单核细胞中的复制

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R. J. Shattock; G. P. Rizzardi; P. Hayes; G. E. Griffin;
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年度 1996
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1. Engagement of adhesion molecules (CD18, CD11a, CD45, CD44, and CD58) enhances human immunodeficiency virus type 1 replication in monocytic cells through a tumor necrosis factor-modulated pathway. [J] . Shattock RJ, Rizzardi GP, Hayes P, The Journal of Infectious Diseases . 1996,第1期

机译：粘附分子（CD18，CD11a，CD45，CD44和CD58）的参与通过肿瘤坏死因子调节途径增强了单核细胞中人免疫缺陷病毒1型的复制。
2. Human Endothelial-Cell Specific Molecule-1 Binds Directly to the Integrin CD11a/CD18 (LFA-1) and Blocks Binding to Intercellular Adhesion Molecule-1 [J] . David Bechard, Arnaud Scherpereel, Hamida Hammad, The Journal of Immunology: Official Journal of the American Association of Immunologists . 2001,第6期

机译：人内皮细胞特异性分子1直接结合到整合素CD11a / CD18（LFA-1），并阻止结合到细胞间粘附分子1。
3. Human Endothelial-Cell Specific Molecule-1 Binds Directly to the Integrin CD11a/CD18 (LFA-1) and Blocks Binding to Intercellular Adhesion Molecule-1 [J] . David Béchard, Arnaud Scherpereel, Hamida Hammad, The journal of immunology . 2001,第6期

机译：人内皮细胞特异性分子1直接结合到整合素CD11a / CD18（LFA-1），并阻止结合到细胞间粘附分子1。
4. Cellular requirements for the budding pathways of avian sarcoma virus and human immunodeficiency virus, type 1. [D] . Pincetic, Andrew. 2009

机译：禽肉瘤病毒和人类免疫缺陷病毒1型的出芽途径对细胞的要求
5. Binding of LFA-1 (CD11a) to Intercellular Adhesion Molecule 3 (ICAM-3; CD50) and ICAM-2 (CD102) Triggers Transmigration of Human Immunodeficiency Virus Type 1-Infected Monocytes through Mucosal Epithelial Cells [O] . Marie-Paule Carreno, Nicolas Chomont, Michel D. Kazatchkine, 2002

机译：LFA-1（CD11a）与细胞间粘附分子3（ICAM-3; CD50）和ICAM-2（CD102）的结合触发人类免疫缺陷病毒1型感染的单核细胞通过粘膜上皮细胞的迁移。
6. Binding of LFA-1 (CD11a) to Intercellular Adhesion Molecule 3 (ICAM-3; CD50) and ICAM-2 (CD102) Triggers Transmigration of Human Immunodeficiency Virus Type 1-Infected Monocytes through Mucosal Epithelial Cells [O] . Carreno, Marie-Paule, Chomont, Nicolas, Kazatchkine, Michel D., 2002

机译：LFA-1（CD11a）与细胞间粘附分子3（ICAM-3; CD50）和ICAM-2（CD102）的结合触发人类免疫缺陷病毒1型感染的单核细胞通过粘膜上皮细胞的迁移。
7. IL-1 and Tumor Necrosis Factor-Alpha Each Up-Regulate Both the Expression of IFN-Gamma Receptors and Enhance IFN-Gamma-Induced HLA-DR Expression on Human Monocytes and a Human Monocytic Cell Line (THP-1) [R] . Krakauer, T., Oppenheim, J. J. 1993

机译：IL-1和肿瘤坏死因子-α各自上调IFN-γ受体的表达并增强人单核细胞和人单核细胞系（THp-1）上IFN-γ诱导的HLa-DR表达

Engagement of Adhesion Molecules (CD18, CD11a, CD45, CD44, and CD58) Enhances Human Immunodeficiency Virus Type 1 Replication in Monocytic Cells through a Tumor Necrosis Factor-Modulated Pathway

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